Evolution of blood ammonia after venipuncture

Groote, J. ; Vandenbroucke, J.
Springer
Published 1958
ISSN:
1573-2568
Source:
Springer Online Journal Archives 1860-2000
Topics:
Medicine
Notes:
Summary The evolution of blood ammonia after venipuncture is studied. Its increase in heparinized blood is small (0.24 µg. in 120 minutes in 1 ml.) but is statistically very significant. This is much more pronounced in oxalated blood. The increase in heparinized blood in cirrhosis is statistically greater than in normals. This occurs also in those cases where the standard value is within 2 σ limits of normal. In hepatic coma with high ammonia it does also exist, but not in those cases with normal blood values. The hypothesis is formulated that an ammoniogenic substance is increased in some pathologic conditions, or that proteins are present which contain more labile amide groups.
Type of Medium:
Electronic Resource
URL:
_version_ 1798296636853583872
autor Groote, J.
Vandenbroucke, J.
autorsonst Groote, J.
Vandenbroucke, J.
book_url http://dx.doi.org/10.1007/BF02231249
datenlieferant nat_lic_papers
hauptsatz hsatz_simple
identnr NLM196996333
issn 1573-2568
journal_name Digestive diseases and sciences
materialart 1
notes Summary The evolution of blood ammonia after venipuncture is studied. Its increase in heparinized blood is small (0.24 µg. in 120 minutes in 1 ml.) but is statistically very significant. This is much more pronounced in oxalated blood. The increase in heparinized blood in cirrhosis is statistically greater than in normals. This occurs also in those cases where the standard value is within 2 σ limits of normal. In hepatic coma with high ammonia it does also exist, but not in those cases with normal blood values. The hypothesis is formulated that an ammoniogenic substance is increased in some pathologic conditions, or that proteins are present which contain more labile amide groups.
package_name Springer
publikationsjahr_anzeige 1958
publikationsjahr_facette 1958
publikationsjahr_intervall 8044:1955-1959
publikationsjahr_sort 1958
publisher Springer
reference 3 (1958), S. 502-510
search_space articles
shingle_author_1 Groote, J.
Vandenbroucke, J.
shingle_author_2 Groote, J.
Vandenbroucke, J.
shingle_author_3 Groote, J.
Vandenbroucke, J.
shingle_author_4 Groote, J.
Vandenbroucke, J.
shingle_catch_all_1 Groote, J.
Vandenbroucke, J.
Evolution of blood ammonia after venipuncture
Summary The evolution of blood ammonia after venipuncture is studied. Its increase in heparinized blood is small (0.24 µg. in 120 minutes in 1 ml.) but is statistically very significant. This is much more pronounced in oxalated blood. The increase in heparinized blood in cirrhosis is statistically greater than in normals. This occurs also in those cases where the standard value is within 2 σ limits of normal. In hepatic coma with high ammonia it does also exist, but not in those cases with normal blood values. The hypothesis is formulated that an ammoniogenic substance is increased in some pathologic conditions, or that proteins are present which contain more labile amide groups.
1573-2568
15732568
Springer
shingle_catch_all_2 Groote, J.
Vandenbroucke, J.
Evolution of blood ammonia after venipuncture
Summary The evolution of blood ammonia after venipuncture is studied. Its increase in heparinized blood is small (0.24 µg. in 120 minutes in 1 ml.) but is statistically very significant. This is much more pronounced in oxalated blood. The increase in heparinized blood in cirrhosis is statistically greater than in normals. This occurs also in those cases where the standard value is within 2 σ limits of normal. In hepatic coma with high ammonia it does also exist, but not in those cases with normal blood values. The hypothesis is formulated that an ammoniogenic substance is increased in some pathologic conditions, or that proteins are present which contain more labile amide groups.
1573-2568
15732568
Springer
shingle_catch_all_3 Groote, J.
Vandenbroucke, J.
Evolution of blood ammonia after venipuncture
Summary The evolution of blood ammonia after venipuncture is studied. Its increase in heparinized blood is small (0.24 µg. in 120 minutes in 1 ml.) but is statistically very significant. This is much more pronounced in oxalated blood. The increase in heparinized blood in cirrhosis is statistically greater than in normals. This occurs also in those cases where the standard value is within 2 σ limits of normal. In hepatic coma with high ammonia it does also exist, but not in those cases with normal blood values. The hypothesis is formulated that an ammoniogenic substance is increased in some pathologic conditions, or that proteins are present which contain more labile amide groups.
1573-2568
15732568
Springer
shingle_catch_all_4 Groote, J.
Vandenbroucke, J.
Evolution of blood ammonia after venipuncture
Summary The evolution of blood ammonia after venipuncture is studied. Its increase in heparinized blood is small (0.24 µg. in 120 minutes in 1 ml.) but is statistically very significant. This is much more pronounced in oxalated blood. The increase in heparinized blood in cirrhosis is statistically greater than in normals. This occurs also in those cases where the standard value is within 2 σ limits of normal. In hepatic coma with high ammonia it does also exist, but not in those cases with normal blood values. The hypothesis is formulated that an ammoniogenic substance is increased in some pathologic conditions, or that proteins are present which contain more labile amide groups.
1573-2568
15732568
Springer
shingle_title_1 Evolution of blood ammonia after venipuncture
shingle_title_2 Evolution of blood ammonia after venipuncture
shingle_title_3 Evolution of blood ammonia after venipuncture
shingle_title_4 Evolution of blood ammonia after venipuncture
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timestamp 2024-05-06T09:55:14.605Z
titel Evolution of blood ammonia after venipuncture
titel_suche Evolution of blood ammonia after venipuncture
topic WW-YZ
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