Arterial oxygen saturation during induction of anaesthesia

THORPE, C. M. ; GAUNTLETT, I. S.

Oxford, UK : Blackwell Publishing Ltd
Published 1990
ISSN:
1365-2044
Source:
Blackwell Publishing Journal Backfiles 1879-2005
Topics:
Medicine
Notes:
Three groups of 10 ASA 1 patients were studied to determine the incidence of hypoxaemia (oxygen saturation ≤ 90%) using pulse oximetry during induction of ‘mask’ anaesthesia, and whether simple oxygenation techniques could prevent its occurrence. We also surveyed all anaesthetists in three major hospitals to ascertain their techniques for this method of anaesthesia. Anaesthesia was induced in all patients with thiopentone and maintained with nitrous oxide and isoflurane. The first group received 33% oxygen in nitrous oxide as carrier gases, a second group a few normal breaths of 100% oxygen during thiopentone administration followed by 33% oxygen in nitrous oxide, while a third group received 100% oxygen after loss of eyelash reflex until spontaneous breathing was established. No patient received positive pressure ventilation before spontaneous breathing was established. Six of the 10 patients in the first group became hypoxaemic compared to none in the second group, and three patients became hypoxaemic in the third group. Thirty-seven percent of anaesthetists who responded to the survey either did not apply positive pressure ventilation before establishment of spontaneous breathing, or only did so if apnoea was prolonged. Only one anaesthetist fully pre-oxygenated patients lungs. We conclude that to avoid the likely occurrence of hypoxaemia during induction of mask anaesthesia, a minimum of a few breaths pre-oxygenation is necessary.
Type of Medium:
Electronic Resource
URL:
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autor THORPE, C. M.
GAUNTLETT, I. S.
book_url http://dx.doi.org/10.1111/j.1365-2044.1990.tb14876.x
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identnr NLZ239171098
insertion_date 2012-04-17
issn 1365-2044
journal_name Anaesthesia
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notes Three groups of 10 ASA 1 patients were studied to determine the incidence of hypoxaemia (oxygen saturation ≤ 90%) using pulse oximetry during induction of ‘mask’ anaesthesia, and whether simple oxygenation techniques could prevent its occurrence. We also surveyed all anaesthetists in three major hospitals to ascertain their techniques for this method of anaesthesia. Anaesthesia was induced in all patients with thiopentone and maintained with nitrous oxide and isoflurane. The first group received 33% oxygen in nitrous oxide as carrier gases, a second group a few normal breaths of 100% oxygen during thiopentone administration followed by 33% oxygen in nitrous oxide, while a third group received 100% oxygen after loss of eyelash reflex until spontaneous breathing was established. No patient received positive pressure ventilation before spontaneous breathing was established. Six of the 10 patients in the first group became hypoxaemic compared to none in the second group, and three patients became hypoxaemic in the third group. Thirty-seven percent of anaesthetists who responded to the survey either did not apply positive pressure ventilation before establishment of spontaneous breathing, or only did so if apnoea was prolonged. Only one anaesthetist fully pre-oxygenated patients lungs. We conclude that to avoid the likely occurrence of hypoxaemia during induction of mask anaesthesia, a minimum of a few breaths pre-oxygenation is necessary.
package_name Blackwell Publishing
publikationsjahr_anzeige 1990
publikationsjahr_facette 1990
publikationsjahr_intervall 8009:1990-1994
publikationsjahr_sort 1990
publikationsort Oxford, UK
publisher Blackwell Publishing Ltd
reference 45 (1990), S. 0
search_space articles
shingle_author_1 THORPE, C. M.
GAUNTLETT, I. S.
shingle_author_2 THORPE, C. M.
GAUNTLETT, I. S.
shingle_author_3 THORPE, C. M.
GAUNTLETT, I. S.
shingle_author_4 THORPE, C. M.
GAUNTLETT, I. S.
shingle_catch_all_1 THORPE, C. M.
GAUNTLETT, I. S.
Arterial oxygen saturation during induction of anaesthesia
Blackwell Publishing Ltd
Three groups of 10 ASA 1 patients were studied to determine the incidence of hypoxaemia (oxygen saturation ≤ 90%) using pulse oximetry during induction of ‘mask’ anaesthesia, and whether simple oxygenation techniques could prevent its occurrence. We also surveyed all anaesthetists in three major hospitals to ascertain their techniques for this method of anaesthesia. Anaesthesia was induced in all patients with thiopentone and maintained with nitrous oxide and isoflurane. The first group received 33% oxygen in nitrous oxide as carrier gases, a second group a few normal breaths of 100% oxygen during thiopentone administration followed by 33% oxygen in nitrous oxide, while a third group received 100% oxygen after loss of eyelash reflex until spontaneous breathing was established. No patient received positive pressure ventilation before spontaneous breathing was established. Six of the 10 patients in the first group became hypoxaemic compared to none in the second group, and three patients became hypoxaemic in the third group. Thirty-seven percent of anaesthetists who responded to the survey either did not apply positive pressure ventilation before establishment of spontaneous breathing, or only did so if apnoea was prolonged. Only one anaesthetist fully pre-oxygenated patients lungs. We conclude that to avoid the likely occurrence of hypoxaemia during induction of mask anaesthesia, a minimum of a few breaths pre-oxygenation is necessary.
1365-2044
13652044
shingle_catch_all_2 THORPE, C. M.
GAUNTLETT, I. S.
Arterial oxygen saturation during induction of anaesthesia
Blackwell Publishing Ltd
Three groups of 10 ASA 1 patients were studied to determine the incidence of hypoxaemia (oxygen saturation ≤ 90%) using pulse oximetry during induction of ‘mask’ anaesthesia, and whether simple oxygenation techniques could prevent its occurrence. We also surveyed all anaesthetists in three major hospitals to ascertain their techniques for this method of anaesthesia. Anaesthesia was induced in all patients with thiopentone and maintained with nitrous oxide and isoflurane. The first group received 33% oxygen in nitrous oxide as carrier gases, a second group a few normal breaths of 100% oxygen during thiopentone administration followed by 33% oxygen in nitrous oxide, while a third group received 100% oxygen after loss of eyelash reflex until spontaneous breathing was established. No patient received positive pressure ventilation before spontaneous breathing was established. Six of the 10 patients in the first group became hypoxaemic compared to none in the second group, and three patients became hypoxaemic in the third group. Thirty-seven percent of anaesthetists who responded to the survey either did not apply positive pressure ventilation before establishment of spontaneous breathing, or only did so if apnoea was prolonged. Only one anaesthetist fully pre-oxygenated patients lungs. We conclude that to avoid the likely occurrence of hypoxaemia during induction of mask anaesthesia, a minimum of a few breaths pre-oxygenation is necessary.
1365-2044
13652044
shingle_catch_all_3 THORPE, C. M.
GAUNTLETT, I. S.
Arterial oxygen saturation during induction of anaesthesia
Blackwell Publishing Ltd
Three groups of 10 ASA 1 patients were studied to determine the incidence of hypoxaemia (oxygen saturation ≤ 90%) using pulse oximetry during induction of ‘mask’ anaesthesia, and whether simple oxygenation techniques could prevent its occurrence. We also surveyed all anaesthetists in three major hospitals to ascertain their techniques for this method of anaesthesia. Anaesthesia was induced in all patients with thiopentone and maintained with nitrous oxide and isoflurane. The first group received 33% oxygen in nitrous oxide as carrier gases, a second group a few normal breaths of 100% oxygen during thiopentone administration followed by 33% oxygen in nitrous oxide, while a third group received 100% oxygen after loss of eyelash reflex until spontaneous breathing was established. No patient received positive pressure ventilation before spontaneous breathing was established. Six of the 10 patients in the first group became hypoxaemic compared to none in the second group, and three patients became hypoxaemic in the third group. Thirty-seven percent of anaesthetists who responded to the survey either did not apply positive pressure ventilation before establishment of spontaneous breathing, or only did so if apnoea was prolonged. Only one anaesthetist fully pre-oxygenated patients lungs. We conclude that to avoid the likely occurrence of hypoxaemia during induction of mask anaesthesia, a minimum of a few breaths pre-oxygenation is necessary.
1365-2044
13652044
shingle_catch_all_4 THORPE, C. M.
GAUNTLETT, I. S.
Arterial oxygen saturation during induction of anaesthesia
Blackwell Publishing Ltd
Three groups of 10 ASA 1 patients were studied to determine the incidence of hypoxaemia (oxygen saturation ≤ 90%) using pulse oximetry during induction of ‘mask’ anaesthesia, and whether simple oxygenation techniques could prevent its occurrence. We also surveyed all anaesthetists in three major hospitals to ascertain their techniques for this method of anaesthesia. Anaesthesia was induced in all patients with thiopentone and maintained with nitrous oxide and isoflurane. The first group received 33% oxygen in nitrous oxide as carrier gases, a second group a few normal breaths of 100% oxygen during thiopentone administration followed by 33% oxygen in nitrous oxide, while a third group received 100% oxygen after loss of eyelash reflex until spontaneous breathing was established. No patient received positive pressure ventilation before spontaneous breathing was established. Six of the 10 patients in the first group became hypoxaemic compared to none in the second group, and three patients became hypoxaemic in the third group. Thirty-seven percent of anaesthetists who responded to the survey either did not apply positive pressure ventilation before establishment of spontaneous breathing, or only did so if apnoea was prolonged. Only one anaesthetist fully pre-oxygenated patients lungs. We conclude that to avoid the likely occurrence of hypoxaemia during induction of mask anaesthesia, a minimum of a few breaths pre-oxygenation is necessary.
1365-2044
13652044
shingle_title_1 Arterial oxygen saturation during induction of anaesthesia
shingle_title_2 Arterial oxygen saturation during induction of anaesthesia
shingle_title_3 Arterial oxygen saturation during induction of anaesthesia
shingle_title_4 Arterial oxygen saturation during induction of anaesthesia
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titel Arterial oxygen saturation during induction of anaesthesia
titel_suche Arterial oxygen saturation during induction of anaesthesia
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