Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission

ISSN:
0022-3956
Source:
Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
Topics:
Medicine
Type of Medium:
Electronic Resource
URL:
_version_ 1798291083003691008
autor Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
autorsonst Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
book_url http://linkinghub.elsevier.com/retrieve/pii/0022-3956(91)90021-2
datenlieferant nat_lic_papers
fussnote Sleep EEG and the nocturnal secretion of cortisol and testosterone in 12 male patients (mean age 46.4+/-11.26 years) with major endogenous depression were investigated concomitantly during acute depression, before treatment and after recovery and drug cessation. Testosterone concentration increased after remission, while cortisol secretion decreased. Sleep EEG disturbances remained unchanged in remitted patients. The data suggest that a blunted testosterone and an elevated cortisol secretion are state markers of acute depression, which normalize independently from sleep structure. An interaction between the hypothalamic-pituitary-gonadal axis and the limbic- hypothalamic-pituitary-adrenocortical axis appears likely.
hauptsatz hsatz_simple
identnr NLZ181860287
issn 0022-3956
journal_name Journal of Psychiatric Research
materialart 1
package_name Elsevier
publikationsort Amsterdam
publisher Elsevier
reference 25 (1991), S. 169-177
search_space articles
shingle_author_1 Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
shingle_author_2 Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
shingle_author_3 Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
shingle_author_4 Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
shingle_catch_all_1 Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
0022-3956
00223956
Elsevier
shingle_catch_all_2 Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
0022-3956
00223956
Elsevier
shingle_catch_all_3 Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
0022-3956
00223956
Elsevier
shingle_catch_all_4 Steiger, A.
von Bardeleben, U.
Wiedemann, K.
Holsboer, F.
Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
0022-3956
00223956
Elsevier
shingle_title_1 Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
shingle_title_2 Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
shingle_title_3 Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
shingle_title_4 Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
sigel_instance_filter dkfz
geomar
wilbert
ipn
albert
fhp
source_archive Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
timestamp 2024-05-06T08:26:58.667Z
titel Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
titel_suche Sleep EEG and nocturnal secretion of testosterone and cortisol in patients with major endogenous depression during acute phase and after remission
Sleep EEG and the nocturnal secretion of cortisol and testosterone in 12 male patients (mean age 46.4+/-11.26 years) with major endogenous depression were investigated concomitantly during acute depression, before treatment and after recovery and drug cessation. Testosterone concentration increased after remission, while cortisol secretion decreased. Sleep EEG disturbances remained unchanged in remitted patients. The data suggest that a blunted testosterone and an elevated cortisol secretion are state markers of acute depression, which normalize independently from sleep structure. An interaction between the hypothalamic-pituitary-gonadal axis and the limbic- hypothalamic-pituitary-adrenocortical axis appears likely.
topic WW-YZ
uid nat_lic_papers_NLZ181860287