Systemic and coronary hemodynamic effects of transthoracic direct current shock
Rowe, G. G. ; Bandow, G. T. ; Laanen, P. J. ; Michler, K. J. ; Zarnstorff, W. C. ; Afonso, S.
Springer
Published 1979
Springer
Published 1979
| ISSN: |
1435-1803
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|---|---|
| Source: |
Springer Online Journal Archives 1860-2000
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| Topics: |
Medicine
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| Notes: |
Summary 1. This study in anesthetized mongrel dogs reports the acute systemic and coronary hemodynamic effects of direct current transthoracic shock similar to that used to change cardiac rhythm. 2. In control animals there was an impressive almost immediate increase in cardiac rate, output and work and an acute transient reduction in systemic, pulmonary and coronary vascular resistance which quickly returned toward normal. 3. The cardiovascular response is damped somewhat but still present after beta adrenergic blockade with propranolol or reserpine depletion of catecholamines. Thus catecholamine release is believed to be partially but not completely responsible for the changes seen. 4. The response is also reduced by vagotomy and by local anesthesia of the chest wall beneath the site of application of the paddles. The possibility is discussed that the response represents the complex reflex known as the “alarm” reaction. 5. The acute hemodynamic changes produced by transthoracic current discharge could be important in some of the infrequent complications of electrical conversion of cardiac arrhythmias. 6. A minor increase in coronary sinus blood temperature occurred with a 200 and 400 Watt second transthoracic discharge. It would seem likely that this should have been greater if a cardiac thermal burning were produced.
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| Type of Medium: |
Electronic Resource
|
| URL: |
| _version_ | 1798296185958563840 |
|---|---|
| autor | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. |
| autorsonst | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. |
| book_url | http://dx.doi.org/10.1007/BF01907683 |
| datenlieferant | nat_lic_papers |
| hauptsatz | hsatz_simple |
| identnr | NLM208619739 |
| issn | 1435-1803 |
| journal_name | Basic research in cardiology |
| materialart | 1 |
| notes | Summary 1. This study in anesthetized mongrel dogs reports the acute systemic and coronary hemodynamic effects of direct current transthoracic shock similar to that used to change cardiac rhythm. 2. In control animals there was an impressive almost immediate increase in cardiac rate, output and work and an acute transient reduction in systemic, pulmonary and coronary vascular resistance which quickly returned toward normal. 3. The cardiovascular response is damped somewhat but still present after beta adrenergic blockade with propranolol or reserpine depletion of catecholamines. Thus catecholamine release is believed to be partially but not completely responsible for the changes seen. 4. The response is also reduced by vagotomy and by local anesthesia of the chest wall beneath the site of application of the paddles. The possibility is discussed that the response represents the complex reflex known as the “alarm” reaction. 5. The acute hemodynamic changes produced by transthoracic current discharge could be important in some of the infrequent complications of electrical conversion of cardiac arrhythmias. 6. A minor increase in coronary sinus blood temperature occurred with a 200 and 400 Watt second transthoracic discharge. It would seem likely that this should have been greater if a cardiac thermal burning were produced. |
| package_name | Springer |
| publikationsjahr_anzeige | 1979 |
| publikationsjahr_facette | 1979 |
| publikationsjahr_intervall | 8024:1975-1979 |
| publikationsjahr_sort | 1979 |
| publisher | Springer |
| reference | 74 (1979), S. 35-45 |
| search_space | articles |
| shingle_author_1 | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. |
| shingle_author_2 | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. |
| shingle_author_3 | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. |
| shingle_author_4 | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. |
| shingle_catch_all_1 | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. Systemic and coronary hemodynamic effects of transthoracic direct current shock Summary 1. This study in anesthetized mongrel dogs reports the acute systemic and coronary hemodynamic effects of direct current transthoracic shock similar to that used to change cardiac rhythm. 2. In control animals there was an impressive almost immediate increase in cardiac rate, output and work and an acute transient reduction in systemic, pulmonary and coronary vascular resistance which quickly returned toward normal. 3. The cardiovascular response is damped somewhat but still present after beta adrenergic blockade with propranolol or reserpine depletion of catecholamines. Thus catecholamine release is believed to be partially but not completely responsible for the changes seen. 4. The response is also reduced by vagotomy and by local anesthesia of the chest wall beneath the site of application of the paddles. The possibility is discussed that the response represents the complex reflex known as the “alarm” reaction. 5. The acute hemodynamic changes produced by transthoracic current discharge could be important in some of the infrequent complications of electrical conversion of cardiac arrhythmias. 6. A minor increase in coronary sinus blood temperature occurred with a 200 and 400 Watt second transthoracic discharge. It would seem likely that this should have been greater if a cardiac thermal burning were produced. 1435-1803 14351803 Springer |
| shingle_catch_all_2 | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. Systemic and coronary hemodynamic effects of transthoracic direct current shock Summary 1. This study in anesthetized mongrel dogs reports the acute systemic and coronary hemodynamic effects of direct current transthoracic shock similar to that used to change cardiac rhythm. 2. In control animals there was an impressive almost immediate increase in cardiac rate, output and work and an acute transient reduction in systemic, pulmonary and coronary vascular resistance which quickly returned toward normal. 3. The cardiovascular response is damped somewhat but still present after beta adrenergic blockade with propranolol or reserpine depletion of catecholamines. Thus catecholamine release is believed to be partially but not completely responsible for the changes seen. 4. The response is also reduced by vagotomy and by local anesthesia of the chest wall beneath the site of application of the paddles. The possibility is discussed that the response represents the complex reflex known as the “alarm” reaction. 5. The acute hemodynamic changes produced by transthoracic current discharge could be important in some of the infrequent complications of electrical conversion of cardiac arrhythmias. 6. A minor increase in coronary sinus blood temperature occurred with a 200 and 400 Watt second transthoracic discharge. It would seem likely that this should have been greater if a cardiac thermal burning were produced. 1435-1803 14351803 Springer |
| shingle_catch_all_3 | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. Systemic and coronary hemodynamic effects of transthoracic direct current shock Summary 1. This study in anesthetized mongrel dogs reports the acute systemic and coronary hemodynamic effects of direct current transthoracic shock similar to that used to change cardiac rhythm. 2. In control animals there was an impressive almost immediate increase in cardiac rate, output and work and an acute transient reduction in systemic, pulmonary and coronary vascular resistance which quickly returned toward normal. 3. The cardiovascular response is damped somewhat but still present after beta adrenergic blockade with propranolol or reserpine depletion of catecholamines. Thus catecholamine release is believed to be partially but not completely responsible for the changes seen. 4. The response is also reduced by vagotomy and by local anesthesia of the chest wall beneath the site of application of the paddles. The possibility is discussed that the response represents the complex reflex known as the “alarm” reaction. 5. The acute hemodynamic changes produced by transthoracic current discharge could be important in some of the infrequent complications of electrical conversion of cardiac arrhythmias. 6. A minor increase in coronary sinus blood temperature occurred with a 200 and 400 Watt second transthoracic discharge. It would seem likely that this should have been greater if a cardiac thermal burning were produced. 1435-1803 14351803 Springer |
| shingle_catch_all_4 | Rowe, G. G. Bandow, G. T. Laanen, P. J. Michler, K. J. Zarnstorff, W. C. Afonso, S. Systemic and coronary hemodynamic effects of transthoracic direct current shock Summary 1. This study in anesthetized mongrel dogs reports the acute systemic and coronary hemodynamic effects of direct current transthoracic shock similar to that used to change cardiac rhythm. 2. In control animals there was an impressive almost immediate increase in cardiac rate, output and work and an acute transient reduction in systemic, pulmonary and coronary vascular resistance which quickly returned toward normal. 3. The cardiovascular response is damped somewhat but still present after beta adrenergic blockade with propranolol or reserpine depletion of catecholamines. Thus catecholamine release is believed to be partially but not completely responsible for the changes seen. 4. The response is also reduced by vagotomy and by local anesthesia of the chest wall beneath the site of application of the paddles. The possibility is discussed that the response represents the complex reflex known as the “alarm” reaction. 5. The acute hemodynamic changes produced by transthoracic current discharge could be important in some of the infrequent complications of electrical conversion of cardiac arrhythmias. 6. A minor increase in coronary sinus blood temperature occurred with a 200 and 400 Watt second transthoracic discharge. It would seem likely that this should have been greater if a cardiac thermal burning were produced. 1435-1803 14351803 Springer |
| shingle_title_1 | Systemic and coronary hemodynamic effects of transthoracic direct current shock |
| shingle_title_2 | Systemic and coronary hemodynamic effects of transthoracic direct current shock |
| shingle_title_3 | Systemic and coronary hemodynamic effects of transthoracic direct current shock |
| shingle_title_4 | Systemic and coronary hemodynamic effects of transthoracic direct current shock |
| sigel_instance_filter | dkfz geomar wilbert ipn albert fhp |
| source_archive | Springer Online Journal Archives 1860-2000 |
| timestamp | 2024-05-06T09:48:05.504Z |
| titel | Systemic and coronary hemodynamic effects of transthoracic direct current shock |
| titel_suche | Systemic and coronary hemodynamic effects of transthoracic direct current shock |
| topic | WW-YZ |
| uid | nat_lic_papers_NLM208619739 |