Incomplete lacunar infarction (Type I b lacunes)

Lammie, G. A. ; Brannan, F. ; Wardlaw, J. M.
Springer
Published 1998
ISSN:
1432-0533
Keywords:
Key words Lacune ; Infarction ; Basal ganglia ; Hypertension ; Embolus
Source:
Springer Online Journal Archives 1860-2000
Topics:
Medicine
Notes:
Abstract The aetiopathogenesis of small, deep (lacunar) infarcts remains controversial. The view that they are caused by occlusive intrinsic small vessel disease is widely held, but is based on only a small number of detailed pathology studies. We describe and illustrate a variant of small, microvessel-associated basal ganglia lesion, the histopathological features of which are distinct from those of classical Types I, II and III lacunes. Their appearances suggest a state of incomplete infarction. The pathogenetic significance of such lesions is discussed, in particular the role of mechanisms causing temporary or only moderately severe ischaemia.
Type of Medium:
Electronic Resource
URL:
http://dx.doi.org/10.1007/s004010050877
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autor Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
autorsonst Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
book_url http://dx.doi.org/10.1007/s004010050877
datenlieferant nat_lic_papers
hauptsatz hsatz_simple
identnr NLM203622596
issn 1432-0533
journal_name Acta neuropathologica
materialart 1
notes Abstract The aetiopathogenesis of small, deep (lacunar) infarcts remains controversial. The view that they are caused by occlusive intrinsic small vessel disease is widely held, but is based on only a small number of detailed pathology studies. We describe and illustrate a variant of small, microvessel-associated basal ganglia lesion, the histopathological features of which are distinct from those of classical Types I, II and III lacunes. Their appearances suggest a state of incomplete infarction. The pathogenetic significance of such lesions is discussed, in particular the role of mechanisms causing temporary or only moderately severe ischaemia.
package_name Springer
publikationsjahr_anzeige 1998
publikationsjahr_facette 1998
publikationsjahr_intervall 8004:1995-1999
publikationsjahr_sort 1998
publisher Springer
reference 96 (1998), S. 163-171
schlagwort Key words Lacune
Infarction
Basal ganglia
Hypertension
Embolus
search_space articles
shingle_author_1 Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
shingle_author_2 Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
shingle_author_3 Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
shingle_author_4 Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
shingle_catch_all_1 Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
Incomplete lacunar infarction (Type I b lacunes)
Key words Lacune
Infarction
Basal ganglia
Hypertension
Embolus
Key words Lacune
Infarction
Basal ganglia
Hypertension
Embolus
Abstract The aetiopathogenesis of small, deep (lacunar) infarcts remains controversial. The view that they are caused by occlusive intrinsic small vessel disease is widely held, but is based on only a small number of detailed pathology studies. We describe and illustrate a variant of small, microvessel-associated basal ganglia lesion, the histopathological features of which are distinct from those of classical Types I, II and III lacunes. Their appearances suggest a state of incomplete infarction. The pathogenetic significance of such lesions is discussed, in particular the role of mechanisms causing temporary or only moderately severe ischaemia.
1432-0533
14320533
Springer
shingle_catch_all_2 Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
Incomplete lacunar infarction (Type I b lacunes)
Key words Lacune
Infarction
Basal ganglia
Hypertension
Embolus
Key words Lacune
Infarction
Basal ganglia
Hypertension
Embolus
Abstract The aetiopathogenesis of small, deep (lacunar) infarcts remains controversial. The view that they are caused by occlusive intrinsic small vessel disease is widely held, but is based on only a small number of detailed pathology studies. We describe and illustrate a variant of small, microvessel-associated basal ganglia lesion, the histopathological features of which are distinct from those of classical Types I, II and III lacunes. Their appearances suggest a state of incomplete infarction. The pathogenetic significance of such lesions is discussed, in particular the role of mechanisms causing temporary or only moderately severe ischaemia.
1432-0533
14320533
Springer
shingle_catch_all_3 Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
Incomplete lacunar infarction (Type I b lacunes)
Key words Lacune
Infarction
Basal ganglia
Hypertension
Embolus
Key words Lacune
Infarction
Basal ganglia
Hypertension
Embolus
Abstract The aetiopathogenesis of small, deep (lacunar) infarcts remains controversial. The view that they are caused by occlusive intrinsic small vessel disease is widely held, but is based on only a small number of detailed pathology studies. We describe and illustrate a variant of small, microvessel-associated basal ganglia lesion, the histopathological features of which are distinct from those of classical Types I, II and III lacunes. Their appearances suggest a state of incomplete infarction. The pathogenetic significance of such lesions is discussed, in particular the role of mechanisms causing temporary or only moderately severe ischaemia.
1432-0533
14320533
Springer
shingle_catch_all_4 Lammie, G. A.
Brannan, F.
Wardlaw, J. M.
Incomplete lacunar infarction (Type I b lacunes)
Key words Lacune
Infarction
Basal ganglia
Hypertension
Embolus
Key words Lacune
Infarction
Basal ganglia
Hypertension
Embolus
Abstract The aetiopathogenesis of small, deep (lacunar) infarcts remains controversial. The view that they are caused by occlusive intrinsic small vessel disease is widely held, but is based on only a small number of detailed pathology studies. We describe and illustrate a variant of small, microvessel-associated basal ganglia lesion, the histopathological features of which are distinct from those of classical Types I, II and III lacunes. Their appearances suggest a state of incomplete infarction. The pathogenetic significance of such lesions is discussed, in particular the role of mechanisms causing temporary or only moderately severe ischaemia.
1432-0533
14320533
Springer
shingle_title_1 Incomplete lacunar infarction (Type I b lacunes)
shingle_title_2 Incomplete lacunar infarction (Type I b lacunes)
shingle_title_3 Incomplete lacunar infarction (Type I b lacunes)
shingle_title_4 Incomplete lacunar infarction (Type I b lacunes)
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source_archive Springer Online Journal Archives 1860-2000
timestamp 2024-05-06T09:35:37.834Z
titel Incomplete lacunar infarction (Type I b lacunes)
titel_suche Incomplete lacunar infarction (Type I b lacunes)
topic WW-YZ
uid nat_lic_papers_NLM203622596