Search Results - (Author, Cooperation:S. Jia)
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1Latest Papers from Table of Contents or Articles in PressRen, W., Liu, Y., Wang, X., Piao, C., Ma, Y., Qiu, S., Jia, L., Chen, B., Wang, Y., Jiang, W., Zheng, S., Liu, C., Dai, N., Lan, F., Zhang, H., Song, W.-c., Du, J.
The American Association of Immunologists (AAI)
Published 2018Staff ViewPublication Date: 2018-02-21Publisher: The American Association of Immunologists (AAI)Print ISSN: 0022-1767Electronic ISSN: 1550-6606Topics: MedicinePublished by: -
2Latest Papers from Table of Contents or Articles in PressLi, T., Song, J., Zhao, X., Yang, Z., Pastel, G., Xu, S., Jia, C., Dai, J., Chen, C., Gong, A., Jiang, F., Yao, Y., Fan, T., Yang, B., Wagberg, L., Yang, R., Hu, L.
American Association for the Advancement of Science (AAAS)
Published 2018Staff ViewPublication Date: 2018-03-12Publisher: American Association for the Advancement of Science (AAAS)Electronic ISSN: 2375-2548Topics: Natural Sciences in GeneralPublished by: -
3Latest Papers from Table of Contents or Articles in PressStaff View
Publication Date: 2018-06-15Publisher: American Physical Society (APS)Print ISSN: 0556-2821Electronic ISSN: 1089-4918Topics: PhysicsKeywords: Particle Physics ExperimentsPublished by: -
4Latest Papers from Table of Contents or Articles in PressS. Y. Xu ; Y. Xia ; L. A. Wray ; S. Jia ; F. Meier ; J. H. Dil ; J. Osterwalder ; B. Slomski ; A. Bansil ; H. Lin ; R. J. Cava ; M. Z. Hasan
American Association for the Advancement of Science (AAAS)
Published 2011Staff ViewPublication Date: 2011-04-02Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsPublished by: -
5Latest Papers from Table of Contents or Articles in PressS. Jia ; Z. Liu ; S. Zhang ; P. Liu ; L. Zhang ; S. H. Lee ; J. Zhang ; S. Signoretti ; M. Loda ; T. M. Roberts ; J. J. Zhao
Nature Publishing Group (NPG)
Published 2016Staff ViewPublication Date: 2016-01-28Publisher: Nature Publishing Group (NPG)Print ISSN: 0028-0836Electronic ISSN: 1476-4687Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsPublished by: -
6Latest Papers from Table of Contents or Articles in PressChen, Y., Li, Z., Han, X., Tian, S., Zhao, J., Chen, F., Su, X., Zhao, J., Zou, Z., Gong, Y., Qu, F., Qiu, G., Wang, S., Jia, X., Lu, Z., Hu, M., Huang, L., Verweij, P. E., Han, L.
The American Society for Microbiology (ASM)
Published 2018Staff ViewPublication Date: 2018-04-27Publisher: The American Society for Microbiology (ASM)Print ISSN: 0066-4804Electronic ISSN: 1098-6596Topics: BiologyMedicinePublished by: -
7Latest Papers from Table of Contents or Articles in PressGuo, Y., Chang, Q., Cheng, L., Xiong, S., Jia, X., Lin, X., Zhao, X.
The American Association of Immunologists (AAI)
Published 2018Staff ViewPublication Date: 2018-10-10Publisher: The American Association of Immunologists (AAI)Print ISSN: 0022-1767Electronic ISSN: 1550-6606Topics: MedicinePublished by: -
8Latest Papers from Table of Contents or Articles in PressS. Y. Xu ; I. Belopolski ; N. Alidoust ; M. Neupane ; G. Bian ; C. Zhang ; R. Sankar ; G. Chang ; Z. Yuan ; C. C. Lee ; S. M. Huang ; H. Zheng ; J. Ma ; D. S. Sanchez ; B. Wang ; A. Bansil ; F. Chou ; P. P. Shibayev ; H. Lin ; S. Jia ; M. Z. Hasan
American Association for the Advancement of Science (AAAS)
Published 2015Staff ViewPublication Date: 2015-07-18Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsPublished by: -
9Latest Papers from Table of Contents or Articles in PressStaff View
Publication Date: 2018-12-01Publisher: American Physical Society (APS)Print ISSN: 0556-2821Electronic ISSN: 1089-4918Topics: PhysicsKeywords: Particle Physics ExperimentsPublished by: -
10Articles: DFG German National LicensesKloeters, O. ; Jia, S. ; Roy, N. ; Leinfellner, G. ; Mustoe, T.A.
Oxford, UK; Malden, USA : Blackwell Publishing Ltd/Inc.
Published 2005Staff ViewISSN: 1524-475XSource: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Introduction: Chronic, nonhealing wounds are often observed in tissues with poor oxygen supply. Impaired reepithelialization is a hallmark of these wounds; however, the pathogenesis of the retarded reepithelialization in chronic, ischemic wounds remains poorly understood. Transforming Growth Factor beta (TGF-beta) is involved in both normal and hypoxic wound healing response and exogenous overexpression of Smad3, a TGF-beta signaling intermediate, has been known to accelerate reepithelialization. In a recent study, Ad-Smad3 injection in the rabbit ear dermal ulcer model showed enhanced reepithelialization and granulation tissue area suggesting a positive effect of Smad3 on wound healing. However, little is known about the role of Smad3 in the ischemic wound healing process. In this study we examined the effect of Smad3 in an ischemic wound model.Methods: Ad-Smad3 or LacZ (108 pfu/wound) empty vector was injected in either ear of White New Zealand Rabbits. Twenty-four hours later, these ears were rendered ischemic using an established model and four 7-mm full-thickness punch wounds were made on each ear.Results: Histological evaluation showed a highly significant increase in reepithelialization, epithelial ingrowth and percentage of epithelialization in Ad-Smad3 transfected wounds versus ischemic wounds transfected with LacZ-empty vector (p 〈 0.01).Conclusion: Our data confirm the enhancing effect of Smad3 on reepithelialization in an ischemic wound model. The deficiency in reepithelialization, as evident in chronic ischemic wounds, could thus be ameliorated by excess Smad3. Therapeutic interventions using overexpressed Smad3 may improve wound healing through accelerated epithelialization in chronic wounds.Type of Medium: Electronic ResourceURL: -
11Articles: DFG German National LicensesProcaccini, P. S. A. ; Jia, S. ; Cross, K. ; Roy, N. ; Mogford, J.E. ; Mustoe, T.A.
Oxford, UK; Malden, USA : Blackwell Science Ltd/Inc.
Published 2004Staff ViewISSN: 1524-475XSource: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Chronic wounds are characterized by a continued inability to heal after an extended period of time (often defined as 3 weeks or longer) and more commonly found in the elderly over areas of compromised blood flow. Collagen is a gene vital to this healing process and Transforming Growth Factor beta 1 (TGFβ1) is an important signaling molecule for collagen induction. We chose to examine the effects of age and ischemia on TGFβ1 regulation using the rat backflap model, which demonstrates negative influences of age and ischemia on the wound healing. Four full-thickness biopsy punches (7 mm) were made centered on the back of aged and young rats. The rostral wound pair was made ischemic by raising a transverse flap (1.8 cm wide × 8 cm length). The caudal wound pair served as a non-ischemic control. Wound contraction was prevented by insertion of a sterilized polyethylene sheet under both the ischemic and nonischemic wounds. RNA was then extracted from wounds at post operation days 3, 7, 10 and 14. The RNA was then reverse transcribed and assayed by Real-Time PCR to examine the relative expression of TGFβ1. TGFβ1 expression in the ischemic wounds of young rats was significantly increased versus nonischemic controls at post-wounding days 3, 7 and 14 (p 〈 0.05, p 〈 0.05, p 〈 0.05, respectively). We also observed a significant increase in TGFβ1 expression in the ischemic of aged animals at day 14 (p = 0.01). The data suggest that there is either a loss of function in TGFβ1’s response to ischemia in the aged animals, or, at least, a significant delay. The results also demonstrate the usefulness of the rat backflap model for the study of age and ischemia on TGFβ1 function in a non-contractile rat wound model.Type of Medium: Electronic ResourceURL: -
12Articles: DFG German National LicensesProcaccini, P. S. A. ; Jia, S. ; Roy, N. ; Mogford, J.E. ; Mustoe, T.A.
Oxford, UK; Malden, USA : Blackwell Science Ltd/Inc.
Published 2004Staff ViewISSN: 1524-475XSource: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Epithelialization and vascularization are key elements involved in the repair of cutaneous wounds. One method of regulation for these pathways is expression of a signaling molecule known as Vascular Endothelial Growth Factor (VEGF), which, in turn, is regulated by the transcription factor Hypoxia-Inducible Factor-1 (HIF-1). It has been suggested that chronic wounds, which are most commonly located in ischemic tissue among the elderly, may be the result of a loss of function in the VEGF regulation pathway. Using our rat back flap model, which demonstrates the negative influences of age and ischemia on wound healing, we examined the relative expression of HIF-1 and VEGF in aged and young rats at 3,7, and 10 days post-wounding. Four full-thickness biopsy punches (7 mm) were made on the back of each aged and young rat. The rostral wound pair was made ischemic by raising a transverse flap (1.8 cm wide × 8 cm length). The caudal wound pair served as a non-ischemic control. Inserting a sterilized polyethylene sheet under both ischemic and nonischemic wounds prevented wound contraction. RNA was extracted from the tissue at the specified time points, and run on Real-Time PCR to determine relative expression of VEGF and HIF-1. Preliminary data suggest that, at the 3 day time point, there is a significant increase in both HIF-1 and VEGF expression in young rats under ischemic conditions and a significant increase in VEGF expression in aged rats under ischemia with a strong (but not significant) trend towards increase in HIF-1 as well. While the rat model does not produce significant differences between aged and young animals, this upregulation under ischemia indicates that HIF-1 and VEGF expression may play a vital role in the early signaling of ischemic wound repair.Type of Medium: Electronic ResourceURL: -
13Articles: DFG German National LicensesVossmeyer, T. ; Jia, S. ; DeIonno, E. ; Diehl, M. R. ; Kim, S.-H.
[S.l.] : American Institute of Physics (AIP)
Published 1998Staff ViewISSN: 1089-7550Source: AIP Digital ArchiveTopics: PhysicsNotes: A scheme for generating complex, spatially separated patterns of multiple types of semiconducting and/or metallic nanocrystals is presented. The process is based on lithographic patterning of organic monolayers that contain a photolabile protection group and are covalently bound to SiO2 surfaces. The process results in spatially and chemically distinct interaction sites on a single substrate. Nanocrystal assembly occurs with a high selectivity on just one type of site. We report on the production of binary, tertiary, and quatemary patterns of nanocrystals. We highlight and discuss the differences between nanocrystal/substrate assembly and molecule/substrate assembly. Finally, we investigate the assembled structures using photoluminescence and absorption spectroscopy. © 1998 American Institute of Physics.Type of Medium: Electronic ResourceURL: -
14Articles: DFG German National LicensesJia, S. ; Mee, R.P. ; Morford, G. ; Agrwal, N. ; Voss, P.G. ; Moutsatsos, I.K. ; Wang, J.L.
Amsterdam : ElsevierStaff ViewISSN: 0378-1119Keywords: Northern blotting ; Recombinant DNA ; cDNA clone ; fusion protein ; immunoblot screening ; lysogen ; peptide mapping ; phage λgt vectorSource: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyType of Medium: Electronic ResourceURL: -
15Articles: DFG German National LicensesStaff View
ISSN: 0921-4534Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: PhysicsType of Medium: Electronic ResourceURL: -
16Articles: DFG German National LicensesStaff View
ISSN: 0921-4534Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: PhysicsType of Medium: Electronic ResourceURL: -
17Articles: DFG German National Licenses
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18Articles: DFG German National LicensesStaff View
ISSN: 1435-1463Keywords: Platelet ; monoamine oxidase ; cytochrome oxidase ; isocitrate dehydrogenase ; Down's syndromeSource: Springer Online Journal Archives 1860-2000Topics: MedicineNotes: Summary Defects in cytochrome oxidase (CO; complex 4) have recently been demonstrated in blood platelets and in brain tissue from patients with Alzheimer's disease (AD) with possible etiological implications. Because of pathogenetic similarities with AD, we have measured the activities of several mitochondrially localised enzymes in the blood platelets of individuals afflicted with trisomy-21 (Down's syndrome). The activities of monoamine oxidase, cytochrome oxidase, isocitrate dehydrogenase, and glutamate dehydrogenase were assayed in washed platelets from sixty caucasian, male and female control individuals (ages 18–60) and ten, young Down's Syndrome patients (ages 9–21). Significant reductions in the activities of monoamine oxidase, cytochrome oxidase, and isocitrate dehydrogenase were found. In all cases the average activities in Down's syndrome individuals were approximately two-thirds those of controls (DS/Controls=0.68, 0.67, 0.64 respectively). The activity of the fourth enzyme studied, glutamate dehydrogenase, was found to be similar to controls. Results suggest that these reductions are a consequence of a generalised mitochondrial disturbance which may lie behind some pathogenetic aspect(s) of the disease.Type of Medium: Electronic ResourceURL: -
19Articles: DFG German National LicensesOuyang, J.-W. ; Liang, H. ; Jia, S.-E. ; Zhang, C. ; Zhao, T.-H. ; He, L.-Z. ; Jia, X.
Amsterdam : ElsevierStaff ViewISSN: 0168-9452Keywords: Anther culture ; Chromosome doubling ; Pollen plant ; WheatSource: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyType of Medium: Electronic ResourceURL: -
20Articles: DFG German National LicensesStaff View
ISSN: 0168-9452Keywords: Japanese morning glory ; Pharbitis nil ; immature embryo culture ; plant regeneration ; somatic embryogenesisSource: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyType of Medium: Electronic ResourceURL: