Search Results - (Author, Cooperation:S. Diggs)
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1Latest Papers from Table of Contents or Articles in PressB. Bolukbasi ; N. Berente ; J. Cutcher-Gershenfeld ; L. Dechurch ; C. Flint ; M. Haberman ; J. L. King ; E. Knight ; B. Lawrence ; E. Masella ; C. McElroy ; B. Mittleman ; M. Nolan ; M. Radik ; N. Shin ; C. A. Thompson ; S. Winter ; I. Zaslavsky ; M. L. Allison ; D. Arctur ; J. Arrigo ; A. K. Aufdenkampe ; J. Bass ; J. Crowell ; M. Daniels ; S. Diggs ; C. Duffy ; Y. Gil ; B. Gomez ; S. Graves ; R. Hazen ; L. Hsu ; D. Kinkade ; K. Lehnert ; C. Marone ; D. Middleton ; A. Noren ; G. Pearthree ; M. Ramamurthy ; E. Robinson ; G. Percivall ; S. Richard ; C. Suarez ; D. Walker
American Association for the Advancement of Science (AAAS)
Published 2013Staff ViewPublication Date: 2013-11-30Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsKeywords: *Access to Information ; Periodicals as Topic/*economics ; Research/*economicsPublished by: -
2Articles: DFG German National LicensesBrayer, J. B. ; Cha, S. ; Nagashima, H. ; Yasunari, U. ; Lindberg, A. ; Diggs, S. ; Martinez, J. ; Goa, J. ; Humphreys-Beher, M. G. ; Peck, A. B.
Oxford, UK : Blackwell Science Ltd
Published 2001Staff ViewISSN: 1365-3083Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: NOD mice manifest many features of autoimmune exocrinopathy (Sjögren's syndrome), a disease generally characterized by a chronic, progressive immunological attack against the exocrine tissues of the salivary and lacrimal glands. Previous studies using the NOD congenic partner strain, NOD.Igμnull, defined an important role for B lymphocytes in the development of xerostomia, implicating autoantibodies reactive with the acetylcholine muscarinic receptor (M3R) as the possible effector mechanism. In the present study, we have examined the impact of the cytokine, interleukin (IL)-4, on autoimmune exocrinopathy by using the IL-4 gene knockout (KO) NOD mouse strain, NOD.IL-4−/−. Despite manifesting the physiological aberrations and marked leukocytic infiltration of the salivary glands characteristic of autoimmune xerostomia in NOD mice, the NOD.IL-4−/− mice do not develop xerostomia. However, NOD.IL-4−/− mice that received adoptively transferred T lymphocytes derived from NOD.Igμ−/− mice progress to xerostomia, thereby reversing the defect. While progression or lack of progression to xerostomia correlated with the ability of the NOD.IL-4−/− mice to express detectable anti-M3R autoantibodies, the precise mechanism of how IL-4 influences the development of autoimmune xerostomia remains speculative.Type of Medium: Electronic ResourceURL: -
3Articles: DFG German National LicensesFormánek, J. ; Kennedy, H. T. ; Korssakow, M. ; Griffin, R. C. ; Woodward, Gl. ; Nikaido, Y. ; Faragher, W. F. ; Morrell, J. C. ; Monroe, G. S. ; Wendt, G. L. ; Diggs, S. H.
Springer
Published 1932Staff ViewISSN: 1618-2650Source: Springer Online Journal Archives 1860-2000Topics: Chemistry and PharmacologyType of Medium: Electronic ResourceURL: -
4Articles: DFG German National LicensesNametkin, S. ; Abakumovsky, L. ; Diggs, S. H. ; Buchler, C. C. ; Henderson, L. M. ; Ferris, S. W. ; Griffith, R. H.
Springer
Published 1930Staff ViewISSN: 1618-2650Source: Springer Online Journal Archives 1860-2000Topics: Chemistry and PharmacologyType of Medium: Electronic ResourceURL: