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1Latest Papers from Table of Contents or Articles in PressSavannah L. Logan, Jacob Thomas, Jinyuan Yan, Ryan P. Baker, Drew S. Shields, Joao B. Xavier, Brian K. Hammer, Raghuveer Parthasarathy
National Academy of Sciences
Published 2018Staff ViewPublication Date: 2018-04-18Publisher: National Academy of SciencesPrint ISSN: 0027-8424Electronic ISSN: 1091-6490Topics: BiologyMedicineNatural Sciences in GeneralPublished by: -
2Latest Papers from Table of Contents or Articles in PressDarton, T. C., Tuyen, H. T., The, H. C., Newton, P. N., Dance, D. A. B., Phetsouvanh, R., Davong, V., Campbell, J. I., Hoang, N. V. M., Thwaites, G. E., Parry, C. M., Thanh, D. P., Baker, S.
The American Society for Microbiology (ASM)
Published 2018Staff ViewPublication Date: 2018-03-28Publisher: The American Society for Microbiology (ASM)Print ISSN: 0066-4804Electronic ISSN: 1098-6596Topics: BiologyMedicinePublished by: -
3Latest Papers from Table of Contents or Articles in PressLiang, L., Cako, A., Urquhart, R., Straus, S. E., Wodchis, W. P., Baker, G. R., Gagliardi, A. R.
BMJ Publishing
Published 2018Staff ViewPublication Date: 2018-01-31Publisher: BMJ PublishingElectronic ISSN: 2044-6055Topics: MedicineKeywords: Open access, Patient-centred medicinePublished by: -
4Latest Papers from Table of Contents or Articles in PressN. Phadnis ; E. P. Baker ; J. C. Cooper ; K. A. Frizzell ; E. Hsieh ; A. F. de la Cruz ; J. Shendure ; J. O. Kitzman ; H. S. Malik
American Association for the Advancement of Science (AAAS)
Published 2015Staff ViewPublication Date: 2015-12-19Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsKeywords: Alleles ; Animals ; Carrier Proteins/genetics/*physiology ; Cell Cycle/*genetics ; Chimera/genetics ; Crosses, Genetic ; Drosophila melanogaster/*genetics/growth & development ; Drosophila simulans/*genetics/growth & development ; Gene Expression Regulation, Developmental ; Genes, Essential/genetics/physiology ; Genes, Insect ; Genes, Lethal/genetics/*physiology ; *Genetic Speciation ; Male ; Molecular Sequence Data ; *Reproductive IsolationPublished by: -
5Latest Papers from Table of Contents or Articles in PressStaff View
Publication Date: 2015-05-15Publisher: Nature Publishing Group (NPG)Print ISSN: 0028-0836Electronic ISSN: 1476-4687Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsKeywords: Animals ; Calcium/metabolism ; Cell Membrane/*enzymology ; Cells, Cultured ; Cytosol/*metabolism ; Drosophila/*enzymology ; Drosophila Proteins/*metabolism ; Membrane Proteins/*metabolism ; Peptide Hydrolases/*metabolism ; ProteolysisPublished by: -
6Latest Papers from Table of Contents or Articles in PressStaff View
Publication Date: 2011-09-23Publisher: Nature Publishing Group (NPG)Print ISSN: 0028-0836Electronic ISSN: 1476-4687Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsKeywords: Academies and Institutes/*organization & administration ; Animals ; Humans ; Military Medicine/*organization & administration ; Pathology/*organization & administration ; United States Government Agencies/*organization & administrationPublished by: -
7Latest Papers from Table of Contents or Articles in PressW. F. Laurance ; D. C. Useche ; J. Rendeiro ; M. Kalka ; C. J. Bradshaw ; S. P. Sloan ; S. G. Laurance ; M. Campbell ; K. Abernethy ; P. Alvarez ; V. Arroyo-Rodriguez ; P. Ashton ; J. Benitez-Malvido ; A. Blom ; K. S. Bobo ; C. H. Cannon ; M. Cao ; R. Carroll ; C. Chapman ; R. Coates ; M. Cords ; F. Danielsen ; B. De Dijn ; E. Dinerstein ; M. A. Donnelly ; D. Edwards ; F. Edwards ; N. Farwig ; P. Fashing ; P. M. Forget ; M. Foster ; G. Gale ; D. Harris ; R. Harrison ; J. Hart ; S. Karpanty ; W. J. Kress ; J. Krishnaswamy ; W. Logsdon ; J. Lovett ; W. Magnusson ; F. Maisels ; A. R. Marshall ; D. McClearn ; D. Mudappa ; M. R. Nielsen ; R. Pearson ; N. Pitman ; J. van der Ploeg ; A. Plumptre ; J. Poulsen ; M. Quesada ; H. Rainey ; D. Robinson ; C. Roetgers ; F. Rovero ; F. Scatena ; C. Schulze ; D. Sheil ; T. Struhsaker ; J. Terborgh ; D. Thomas ; R. Timm ; J. N. Urbina-Cardona ; K. Vasudevan ; S. J. Wright ; G. J. Arias ; L. Arroyo ; M. Ashton ; P. Auzel ; D. Babaasa ; F. Babweteera ; P. Baker ; O. Banki ; M. Bass ; I. Bila-Isia ; S. Blake ; W. Brockelman ; N. Brokaw ; C. A. Bruhl ; S. Bunyavejchewin ; J. T. Chao ; J. Chave ; R. Chellam ; C. J. Clark ; J. Clavijo ; R. Congdon ; R. Corlett ; H. S. Dattaraja ; C. Dave ; G. Davies ; M. Beisiegel Bde ; N. da Silva Rde ; A. Di Fiore ; A. Diesmos ; R. Dirzo ; D. Doran-Sheehy ; M. Eaton ; L. Emmons ; A. Estrada ; C. Ewango ; L. Fedigan ; F. Feer ; B. Fruth ; J. G. Willis ; U. Goodale ; S. Goodman ; J. C. Guix ; P. Guthiga ; W. Haber ; K. Hamer ; I. Herbinger ; J. Hill ; Z. Huang ; I. F. Sun ; K. Ickes ; A. Itoh ; N. Ivanauskas ; B. Jackes ; J. Janovec ; D. Janzen ; M. Jiangming ; C. Jin ; T. Jones ; H. Justiniano ; E. Kalko ; A. Kasangaki ; T. Killeen ; H. B. King ; E. Klop ; C. Knott ; I. Kone ; E. Kudavidanage ; J. L. Ribeiro ; J. Lattke ; R. Laval ; R. Lawton ; M. Leal ; M. Leighton ; M. Lentino ; C. Leonel ; J. Lindsell ; L. Ling-Ling ; K. E. Linsenmair ; E. Losos ; A. Lugo ; J. Lwanga ; A. L. Mack ; M. Martins ; W. S. McGraw ; R. McNab ; L. Montag ; J. M. Thompson ; J. Nabe-Nielsen ; M. Nakagawa ; S. Nepal ; M. Norconk ; V. Novotny ; S. O'Donnell ; M. Opiang ; P. Ouboter ; K. Parker ; N. Parthasarathy ; K. Pisciotta ; D. Prawiradilaga ; C. Pringle ; S. Rajathurai ; U. Reichard ; G. Reinartz ; K. Renton ; G. Reynolds ; V. Reynolds ; E. Riley ; M. O. Rodel ; J. Rothman ; P. Round ; S. Sakai ; T. Sanaiotti ; T. Savini ; G. Schaab ; J. Seidensticker ; A. Siaka ; M. R. Silman ; T. B. Smith ; S. S. de Almeida ; N. Sodhi ; C. Stanford ; K. Stewart ; E. Stokes ; K. E. Stoner ; R. Sukumar ; M. Surbeck ; M. Tobler ; T. Tscharntke ; A. Turkalo ; G. Umapathy ; M. van Weerd ; J. V. Rivera ; M. Venkataraman ; L. Venn ; C. Verea ; C. V. de Castilho ; M. Waltert ; B. Wang ; D. Watts ; W. Weber ; P. West ; D. Whitacre ; K. Whitney ; D. Wilkie ; S. Williams ; D. D. Wright ; P. Wright ; L. Xiankai ; P. Yonzon ; F. Zamzani
Nature Publishing Group (NPG)
Published 2012Staff ViewPublication Date: 2012-07-27Publisher: Nature Publishing Group (NPG)Print ISSN: 0028-0836Electronic ISSN: 1476-4687Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsKeywords: Agriculture/statistics & numerical data ; Animals ; *Biodiversity ; Conservation of Natural Resources/*statistics & numerical data ; Data Collection ; Ecology/statistics & numerical data ; Endangered Species/*statistics & numerical data ; Environmental Pollution/adverse effects/statistics & numerical data ; Fires/statistics & numerical data ; Forestry/statistics & numerical data ; Interviews as Topic ; Mining/statistics & numerical data ; Population Growth ; Rain ; Reproducibility of Results ; Research Personnel ; Surveys and Questionnaires ; Temperature ; Trees/*physiology ; *Tropical ClimatePublished by: -
8Articles: DFG German National LicensesWe, Hoy ; P, Baker ; Z, Wang ; A, Cass ; Jd, Mathews ; P., Van Buynder
Melbourne, Australia : Blackwell Science Pty
Published 2000Staff ViewISSN: 1440-1797Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Background: Chronic disease programs are poorly developed in most Aboriginal communities. Much disease is unrecognised or inadequately treated, although appropriate interventions profoundly reduce morbidity and mortality in nonAboriginal populations. Programs of improved management must aspire to best practice for all, so that maintaining parallel untreated control groups is unethical. This poses challenges for evaluating effect.Methods: We identified a large burden of chronic disease in a 1990-1995 screening program in one community, and started a renal & cardiovascular-protection program in Nov 1995. This centred around use of ACE inhibitors, rigorous BP control, better control of glycemia and lipids, & health education. By late 1999 about 275 people, or 30% of all adults had enrolled. The courses of BP, albuminuria and GFR was compared with those in the pre-program era (ANZSN, 1999). Treatment effects on renal failure & natural death were estimated in 3 ways. 1) Comparison of these endpoints in the “intention to treat” group with those in persons potentially eligible for treatment on their 1990-1995 screening results, ‘controls’. There was 50% overlap between the groups, & controls were younger and had less severe disease than the treatment group. 2.Community-based trends in endpoints. 3. Comparison of these trends with those in other NT Top End communities.Results: 1. Risk ratios of rates, Kaplan Meier survivals, and Cox hazard ratios all showed better survival of the treated group over controls, with estimates of 41%-64% reductions in endpoints, after accounting for disease severity. 2. Dialysis starts in the entire community have fallen by at least 38% and natural deaths by 32%. 3. In contrast dialysis continue to increase at 11% per yr in other communities and deaths have not fallen. These results all suggest a marked benefit from the treatment program. Similar methods might be used where truly controlled observations are not feasible.Type of Medium: Electronic ResourceURL: -
9Articles: DFG German National LicensesWe, Hoy ; P, Baker ; Z, Wang ; A, Cass ; Jd, Mathews ; P., Van Buynder
Melbourne, Australia : Blackwell Science Pty
Published 2000Staff ViewISSN: 1440-1797Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Background: Chronic disease programs are poorly developed in most Aboriginal communities. Much disease is unrecognised or inadequately treated, although appropriate interventions profoundly reduce morbidity and mortality in nonAboriginal populations. Programs of improved management must aspire to best practice for all, so that maintaining parallel untreated control groups is unethical. This poses challenges for evaluating effect.Methods: We identified a large burden of chronic disease in a 1990-1995 screening program in one community, and started a renal & cardiovascular-protection program in Nov 1995. This centred around use of ACE inhibitors, rigorous BP control, better control of glycemia and lipids, & health education. By late 1999 about 275 people, or 30% of all adults had enrolled. The courses of BP, albuminuria and GFR was compared with those in the pre-program era (ANZSN, 1999). Treatment effects on renal failure & natural death were estimated in 3 ways. 1) Comparison of these endpoints in the “intention to treat” group with those in persons potentially eligible for treatment on their 1990-1995 screening results, ‘controls’. There was 50% overlap between the groups, & controls were younger and had less severe disease than the treatment group. 2.Community-based trends in endpoints. 3. Comparison of these trends with those in other NT Top End communities.Results: 1. Risk ratios of rates, Kaplan Meier survivals, and Cox hazard ratios all showed better survival of the treated group over controls, with estimates of 41%-64% reductions in endpoints, after accounting for disease severity. 2. Dialysis starts in the entire community have fallen by at least 38% and natural deaths by 32%. 3. In contrast dialysis continue to increase at 11% per yr in other communities and deaths have not fallen. These results all suggest a marked benefit from the treatment program. Similar methods might be used where truly controlled observations are not feasible.Type of Medium: Electronic ResourceURL: -
10Articles: DFG German National LicensesWe, Hoy ; Z, Wang ; P, Baker ; S, McDonald ; Jd, Mathews ; P., Van Buynder
Melbourne, Australia : Blackwell Science Pty
Published 2000Staff ViewISSN: 1440-1797Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Background: Adult death rates of A in the NT are increased 3-6-fold. Albuminuria (high albumin/creatinine ratio, ACR, gm/mol) is also pervasive & progressive. In a study of one community, ACR correlated with indices of poor health, including early malnutrition, increasing BP, glycemia and dyslipidemia, heavy drinking, & infections, and it predicts not only renal failure, but natural death (McDonald et al CEPP 1999). We further develop this last association and discuss its implications.Methods: 763 adults (20+ yr)were screened & followed for 1-8 yr (mean 4.3) yr, until death, renal failure, start of treatment or close-out in April ‘98.Results: Twelve people started dialysis: all had ACR 100+ at baseline. There were 61 natural deaths; 21 were cardiovascular (CV), 40 others were due to lung disease, infections, liver disease, amyloidosis, cancer etc. Death rates (CV and nonCV) correlated with increasing baseline ACR over a continuum in nondiabetics and diabetics. People with baseline ACR 〈 3.4 had a death rate of 4.7./1000 person yr, the referent group, while hazard ratios for natural death of persons with baseline ACRs of 3.4-33, 34-99 and 100+ were 2.3 (95% CI 0.98-5.3), 3.2 (1.3-7.9) and 5.1(2.1-12.8) respectively (p = .0029) after accounting for age and sex. The population attributable risk for natural death or dialysis marked by ACR 3.4+ was 0.67 (CI .37-.83), and for natural death alone was 0.59 (.22-.79) after accounting for age, sex, diabetes & hyper-tension. The excess CV mortality of diabetics was entirely represented by their higher baseline ACRs.Conclusion:. Much of the excess mortality in this group is predicted by albumin-uria. Thus the kidney, whose maturation is very sensitive to early environment, and has vast vascular networks, high blood flow, high metabolic rate, and concentrating, phagocytic & endocrine functions, seems an exquisite marker of general health in A people. The ACR is a simple, reproducible, cheap test, which has wide potential application in definition of community health profiles and trends, and in defining diagnosis and prognosis. Initiation and titration of treatment might be guided by the ACR, and arrest of its progression results in dramatic reductions in natural deaths and renal failure.(Hoy et al, ANZSN '99).Type of Medium: Electronic ResourceURL: -
11Latest Papers from Table of Contents or Articles in PressLarry E. Taylor IIBrandon C. Knott; John O. Baker; P. Markus Alahuhta; Sarah E. Hobdey; Jeffrey G. Linger; Vladimir V. Lunin; Antonella Amore; Venkataramanan Subramanian; Kara Podkaminer; Qi Xu; Todd A. Vander; Wall; Logan A. Schuster; Yogesh B. Chaudhari; William S. Adney; Michael F. Crowley; Michael E. Himmel; Stephen R. Decker; Gregg T. Beckham
Nature Publishing Group (NPG)
Published 2018Staff ViewPublication Date: 2018-03-23Publisher: Nature Publishing Group (NPG)Electronic ISSN: 2041-1723Topics: BiologyChemistry and PharmacologyNatural Sciences in GeneralPhysicsPublished by: -
12Latest Papers from Table of Contents or Articles in PressTong, C., Wen, L., Xia, Y., Leong, P., Wang, L., Fan, X., Han, T.-l., Craig, J. M., Baker, P., Saffery, R., Qi, H.
BMJ Publishing
Published 2018Staff ViewPublication Date: 2018-02-23Publisher: BMJ PublishingElectronic ISSN: 2044-6055Topics: MedicineKeywords: Open access, PaediatricsPublished by: -
13Latest Papers from Table of Contents or Articles in PressC. Gauvin-Ndiaye, T. E. Baker, P. Karan, É. Massé, M. Balli, N. Brahiti, M. A. Eskandari, P. Fournier, A.-M. S. Tremblay, and R. Nourafkan
American Physical Society (APS)
Published 2018Staff ViewPublication Date: 2018-09-18Publisher: American Physical Society (APS)Print ISSN: 1098-0121Electronic ISSN: 1095-3795Topics: PhysicsKeywords: Electronic structure and strongly correlated systemsPublished by: -
14Articles: DFG German National LicensesStaff View
ISSN: 1471-0528Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineType of Medium: Electronic ResourceURL: -
15Articles: DFG German National LicensesStaff View
ISSN: 1471-4159Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Abstract: The uptake of glutamate and other acidic amino acids into barnacle single muscle fibres has been characterized. The uptake of glutamate consists of two components, one Na-independent and one Na-dependent. The Na-dependent uptake is saturable (half-maximal at 250 μM external glutamate) and is inhibited by a variety of analogues of which L-cysteate and D- and L-aspartate are the most potent. These amino acids are also transported into the muscle in a Na-dependent manner. The excitatory agonists kainate, quisqualate, and N-methyl-D-aspartate do not inhibit or affect uptake in any way. Progressive replacement of external Na by choline reduces uptake with very little effect on the apparent affinity for glutamate, suggesting that Na and glutamate bind to the transporter independently. The kinetics of activation are consistent with a requirement for at least two Na ions. Na activation of glutamate uptake can be inhibited by guanidinium with kinetics that are consistent with competitive inhibition at the Na binding site. Studies on the efflux of L-glutamate and other analogues have shown that efflux rates are only slightly increased by the removal of Na and do not seem to be affected in any clear manner by external levels of acidic amino acids.Type of Medium: Electronic ResourceURL: -
16Articles: DFG German National LicensesSteegers-Theunissen, R. P. M. ; Smith, S. C. ; Steegers, E. A. P. ; Guilbert, L. J. ; Baker, P. N.
Oxford, UK : Blackwell Publishing Ltd
Published 2000Staff ViewISSN: 1471-0528Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Effects of folate deficiency on the rate of apoptosis in human cytotrophoblastic cells has been investigated. Apoptosis was determined in cytotrophoblastic cells after culture in 1. control medium, 2. folate-free medium and 3. folate-free medium plus 10% fetal calf serum. Apoptosis rates in cells cultured in mediums 2 and 3 were significantly higher than those cultured in the control medium (P 〈 0.02 and P 〈 0.03, respectively). In conclusion, human cytotrophoblastic cells show a significantly increased rate of apoptosis in vitro after culture in a folate-free medium. Possible explanations for the association between folate deficiency and pregnancy complications are suggested.Type of Medium: Electronic ResourceURL: -
17Articles: DFG German National LicensesStaff View
ISSN: 1471-0528Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Summary. The outcome of 140 pregnancies complicated by unexplained abdominal pain was compared with that in a comparison group of 280 women who gave birth at the same time. The two groups were comparable for maternal age and parity but the study group contained a significantly higher proportion of smokers, unmarried women, and women whose partners were unemployed (P〈0·001). All the pregnancies resulted in livebirths. There were no statistically significant differences in birthweight, gestational age at delivery or mode of delivery between the two groups.Type of Medium: Electronic ResourceURL: -
18Articles: DFG German National LicensesCrocker, I. P. ; Wellings, R. P. ; Fletcher, J. ; Baker, P. N.
Oxford, UK : Blackwell Publishing Ltd
Published 1999Staff ViewISSN: 1471-0528Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Objective To investigate the function of neutrophils in normal pregnancy and pre-eclampsia.Design Baseline levels and activated responses of peripheral blood neutrophils were measured in response to the physiological agonists, n-formyl-met-leu-phe (fMLP) and zymosan activated serum.Sample Neutrophils of 16 pre-eclamptic, 17 normal pregnant (third trimester) and 15 nonpregnant age-matched control women were calculated.Setting Antenatal Clinic, City Hospital, Nottingham.Methods Neutrophil superoxide anion production was determined by lucigenin-enhanced chemilumi-nescence; the release of secondary granule lactoferrin by ELISA; and the expression of cell surface adhesion molecules CD1 1b, CD 18 and L-selectin (CD62L) by flow cytometric analysis.Results Superoxide anion generation was reduced in the pregnant group compared with nonpregnant controls [fMLP by 51 % (P= 0.03) and zymosan activated serum by 56% (P= 〉 0.01)] but pre-eclamptic measurements did not show a similar reduction. There were no differences between the three study groups in the plasma levels of lactofenin, or in the stimulated expression and release of CD1 Ib and CD18, or lactoferrin and L-selectin. The base-line levels for the production of superoxide anions; the expression of CD11b or CD 18 or L-selectin; and the release of lactofemn showed no significant differences.Conclusions Circulating neutrophils in pregnancy and pre-eclampsia are neither activated nor primed in vivo, however the release of reactive oxygen species is diminished in normal pregnancy. In comparison, an elevation of reactive oxygen generation in pre-eclampsia may highlight a role for neutrophils in the oxidative stress and pathophysiology of this disease.Type of Medium: Electronic ResourceURL: -
19Articles: DFG German National LicensesMorgan, L. ; Crawshaw, S. ; Baker, P. N. ; Pipkin, F. Broughton ; Kalsheker, N.
Oxford, UK : Blackwell Publishing Ltd
Published 1999Staff ViewISSN: 1471-0528Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Objective To compare the angiotensinogen genotypes in normotensive and pre-eclamptic pregnancies in maternal and fetal samples.Design Prospective observational study.Setting University Hospital, Queen's Medical Centre, Nottingham.Population Forty-three women with pre-eclampsia and 84 normotensive pregnant women. Fetal samples were available for genotyping from 96% of the pregnancies.Methods Maternal and fetal DNA was genotyped at angiotensinogen codon 235 and at a dinucleotide repeat polymorphism in the 3′ flanking region of the gene. Angiotensinogen and renin concentrations were measured in maternal plasma by radioimmunoassay.Results In contrast to earlier studies, no association was demonstrated between the angiotensinogen 235 Thr variant and pre-eclampsia. Normotensive pregnant women homozygous for this variant had significantly lower plasma angiotensinogen concentrations (median 2.2 ng AI/mL; IQR 1.8–3.0) than women homozygous for the 235 Met allele (3.6 ng AI/mL; IQR 2.5–4.1; P= 0.04). In pre-eclamptic pregnancies, 79% (11/14) of mothers heterozygous for the dinucleotide repeat allele designated A9 transmitted this allele to the fetus, more frequently than would be expected by chance (P= 0.02). The A9 allele was associated with low plasma angiotensinogen concentrations (P= 0.001) and high renin concentrations (P= 0.02) in normotensive women.Conclusions There is no evidence that the angiotensinogen 235 Thr allele is associated with pre-eclampsia in the Nottingham population. The angiotensinogen 235 Thr allele is associated with low plasma angiotensinogen concentrations in normotensive pregnant women, in contrast to the high levels associated with this variant in non-pregnant women, suggesting that regulation of angiotensinogen expression in normal pregnancy may differ significantly from that in the non-pregnant state. There is preliminary evidence that maternal-fetal transmission of an angiotensinogen allele associated with low plasma angiotensinogen concentrations is associated with pre-eclampsia. Impaired generation of angiotensin II at the maternal-fetal interface may be a factor in the pathogenesis of pre-eclampsia.Type of Medium: Electronic ResourceURL: -
20Articles: DFG German National LicensesHayman, R ; Brocklesby, J ; Warren, A ; Ashworth, J ; Johnson, I ; Baker, P.
Oxford, UK : Blackwell Publishing Ltd
Published 1998Staff ViewISSN: 1471-0528Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineType of Medium: Electronic ResourceURL: