Search Results - (Author, Cooperation:L. Pillus)

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  1. 1
    H. Basnet ; X. B. Su ; Y. Tan ; J. Meisenhelder ; D. Merkurjev ; K. A. Ohgi ; T. Hunter ; L. Pillus ; M. G. Rosenfeld
    Nature Publishing Group (NPG)
    Published 2014
    Staff View
    Publication Date:
    2014-09-26
    Publisher:
    Nature Publishing Group (NPG)
    Print ISSN:
    0028-0836
    Electronic ISSN:
    1476-4687
    Topics:
    Biology
    Chemistry and Pharmacology
    Medicine
    Natural Sciences in General
    Physics
    Keywords:
    Amino Acid Sequence ; Casein Kinase II/*metabolism ; Cell Line ; Conserved Sequence ; Histones/*chemistry/genetics/*metabolism ; Humans ; Molecular Sequence Data ; Phosphorylation ; Saccharomyces cerevisiae/genetics/metabolism ; *Transcription Elongation, Genetic ; Tyrosine/chemistry/*metabolism ; Ubiquitination/genetics
    Published by:
    Latest Papers from Table of Contents or Articles in Press
  2. 2
    Pillus, L.

    Amsterdam : Elsevier
    Staff View
    ISSN:
    0955-0674
    Source:
    Elsevier Journal Backfiles on ScienceDirect 1907 - 2002
    Topics:
    Biology
    Medicine
    Type of Medium:
    Electronic Resource
    URL:
    Articles: DFG German National Licenses
  3. 3
    Lowell, J. E. ; Pillus, L.
    Springer
    Published 1998
    Staff View
    ISSN:
    1420-9071
    Keywords:
    Key words.Saccharomyces cerevisiae; telomeres; silencing; chromatin; telomerase; recombination.
    Source:
    Springer Online Journal Archives 1860-2000
    Topics:
    Biology
    Medicine
    Notes:
    Abstract. Unusual chromatin structures underlie epigenetic effects at the silent mating-type loci and telomeres in yeast. Many of the same genes appear to function in transcriptional silencing observed at both the silent mating-type loci and at telomeres. The observation that these loci are united by a requirement for shared factors suggests that the structure of chromatin at these regions is similar. Alteration of telomeric chromatin components affects regulation of transcription, telomeric length, recombination and chromosomal stability. Mutations in TLC1 and EST2, which both encode components of telomerase, cause identical phenotypes progressive shortening of telomeric DNA, increased chromosome loss and eventually cell death. In this review, we examine the relationship between telomeric chromatin and telomere replication and discuss the possibility that telomerase itself is an integral part of telomeric chromatin structure.
    Type of Medium:
    Electronic Resource
    URL:
    Articles: DFG German National Licenses