Search Results - (Author, Cooperation:J. Langhorne)
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1Latest Papers from Table of Contents or Articles in PressP. Wongpan, K. M. Meiners, P. J. Langhorne, P. Heil, I. J. Smith, G. H. Leonard, R. A. Massom, L. A. Clementson, T. G. Haskell
Wiley-Blackwell
Published 2018Staff ViewPublication Date: 2018-02-24Publisher: Wiley-BlackwellPrint ISSN: 0148-0227Topics: GeosciencesPhysicsPublished by: -
2Latest Papers from Table of Contents or Articles in PressP. J. Spence ; W. Jarra ; P. Levy ; A. J. Reid ; L. Chappell ; T. Brugat ; M. Sanders ; M. Berriman ; J. Langhorne
Nature Publishing Group (NPG)
Published 2013Staff ViewPublication Date: 2013-05-31Publisher: Nature Publishing Group (NPG)Print ISSN: 0028-0836Electronic ISSN: 1476-4687Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsKeywords: Animals ; Culicidae/*parasitology ; Erythrocytes/parasitology ; Host-Parasite Interactions/*immunology ; Insect Vectors/*parasitology ; Malaria/immunology/parasitology/transmission ; Malaria Vaccines/immunology ; Mice ; Mice, Inbred C57BL ; Plasmodium chabaudi/growth & development/*immunology/isolation & ; purification/*pathogenicity ; Serial Passage ; Virulence/immunologyPublished by: -
3Articles: DFG German National LicensesStaff View
ISSN: 1365-3083Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Medial histocompatibility (H) antigens are weak H antigens, recognized by unrestricted T cells; they differ thus from both major and minor H antigens. An example, Qed-1, is described in detail, and other known medial H antigens of the mouse are reviewed. The structural and genetic relationships of major and medial H antigens and their role in T cell recognition are discussed.Type of Medium: Electronic ResourceURL: -
4Articles: DFG German National LicensesStaff View
ISSN: 0169-4758Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyMedicineType of Medium: Electronic ResourceURL: -
5Articles: DFG German National LicensesStaff View
ISSN: 0169-4758Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyMedicineType of Medium: Electronic ResourceURL: -
6Articles: DFG German National LicensesStaff View
ISSN: 1365-3083Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: T cells from spleens of mice infected with the erythrocytic stages of Plasmodium chabaudi chabaudi have been analysed with respect to their expression of surface molecules CD3, CD4 and CDS and T-cell receptor (TCR)αβ and γδ. The majority of T cells from infected mice were αβTCR +. However, there was an increase of approximately 8–10-fold in the proportion and total number of γδ T cells. Immunocytochemical analysis of sections of spleens taken from infected C57BL/6 mice during a primary infection showed that this increase took place particularly in the non-lymphoid areas. Within the αβ TCR+ T-cell population, both CD4+ T cells and CD8+ T cells were represented in proportions similar to those observed in normal uninfected mice. Stimulation of splenic T cells from infected mice with P. chabaudi-infected erythrocytes in vitro resulted ina blasted cell population composed predominantly of αβTTCR+ T cells with no preferential expansion of γβTCR+ T cells. There was no evidence of superantigen-like stimulation of T cells bearing particular Vβ chains of the TCR. The representation of the different Vβ chains within the population was not significantly different from that seen in uninfected mice.Type of Medium: Electronic ResourceURL: