Search Results - (Author, Cooperation:J. Gordon)
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1Printed BooksStaff View Availability
Person(s): Raitt, J. GordonType of Medium: UnknownPages: 287 S. : Ill., graph. Darst.Language: German -
2Latest Papers from Table of Contents or Articles in PressStaff View
Publication Date: 2018-07-05Publisher: Royal SocietyElectronic ISSN: 2054-5703Topics: Natural Sciences in GeneralKeywords: chemical biologyPublished by: -
3Latest Papers from Table of Contents or Articles in PressShah, M. A., Starodub, A., Sharma, S., Berlin, J., Patel, M., Wainberg, Z. A., Chaves, J., Gordon, M., Windsor, K., Brachmann, C. B., Huang, X., Vosganian, G., Maltzman, J. D., Smith, V., Silverman, J. A., Lenz, H.-J., Bendell, J. C.
The American Association for Cancer Research (AACR)
Published 2018Staff ViewPublication Date: 2018-08-16Publisher: The American Association for Cancer Research (AACR)Print ISSN: 1078-0432Electronic ISSN: 1557-3265Topics: MedicinePublished by: -
4Latest Papers from Table of Contents or Articles in PressHang Thi Phan, Hang Thi Thuy Tran, Hanh Thi My Tran, Anh Pham Phuong Dinh, Ha Thanh Ngo, Jenny Theorell-Haglow and Christopher J. Gordon
BioMed Central
Published 2018Staff ViewPublication Date: 2018-03-09Publisher: BioMed CentralElectronic ISSN: 1471-2334Topics: MedicinePublished by: -
5Latest Papers from Table of Contents or Articles in PressCraig J. Anderson, John G. Oakeshott, Wee Tek Tay, Karl H. J. Gordon, Andreas Zwick, Tom K. Walsh
National Academy of Sciences
Published 2018Staff ViewPublication Date: 2018-05-09Publisher: National Academy of SciencesPrint ISSN: 0027-8424Electronic ISSN: 1091-6490Topics: BiologyMedicineNatural Sciences in GeneralPublished by: -
6Latest Papers from Table of Contents or Articles in PressKaplanis, J., Gordon, A., Shor, T., Weissbrod, O., Geiger, D., Wahl, M., Gershovits, M., Markus, B., Sheikh, M., Gymrek, M., Bhatia, G., Mac; Arthur, D. G., Price, A. L., Erlich, Y.
American Association for the Advancement of Science (AAAS)
Published 2018Staff ViewPublication Date: 2018-04-13Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyGeosciencesComputer ScienceMedicineNatural Sciences in GeneralPhysicsKeywords: GeneticsPublished by: -
7Latest Papers from Table of Contents or Articles in PressRobert P. Law, Stephen J. Atkinson, Paul Bamborough, Chun-wa Chung, Emmanuel H. Demont, Laurie J. Gordon, Matthew Lindon, Rab K. Prinjha, Allan J. B. Watson, David J. Hirst
American Chemical Society (ACS)
Published 2018Staff ViewPublication Date: 2018-05-05Publisher: American Chemical Society (ACS)Topics: Chemistry and PharmacologyPublished by: -
8Latest Papers from Table of Contents or Articles in PressJ. Peebles, A. V. Arefiev, S. Zhang, C. McGuffey, M. Spinks, J. Gordon, E. W. Gaul, G. Dyer, M. Martinez, M. E. Donovan, T. Ditmire, J. Park, H. Chen, H. S. McLean, M. S. Wei, S. I. Krasheninnikov, and F. N. Beg
American Physical Society (APS)
Published 2018Staff ViewPublication Date: 2018-11-08Publisher: American Physical Society (APS)Print ISSN: 1539-3755Electronic ISSN: 1550-2376Topics: PhysicsKeywords: Plasma PhysicsPublished by: -
9Latest Papers from Table of Contents or Articles in PressR. A. Seder ; L. J. Chang ; M. E. Enama ; K. L. Zephir ; U. N. Sarwar ; I. J. Gordon ; L. A. Holman ; E. R. James ; P. F. Billingsley ; A. Gunasekera ; A. Richman ; S. Chakravarty ; A. Manoj ; S. Velmurugan ; M. Li ; A. J. Ruben ; T. Li ; A. G. Eappen ; R. E. Stafford ; S. H. Plummer ; C. S. Hendel ; L. Novik ; P. J. Costner ; F. H. Mendoza ; J. G. Saunders ; M. C. Nason ; J. H. Richardson ; J. Murphy ; S. A. Davidson ; T. L. Richie ; M. Sedegah ; A. Sutamihardja ; G. A. Fahle ; K. E. Lyke ; M. B. Laurens ; M. Roederer ; K. Tewari ; J. E. Epstein ; B. K. Sim ; J. E. Ledgerwood ; B. S. Graham ; S. L. Hoffman
American Association for the Advancement of Science (AAAS)
Published 2013Staff ViewPublication Date: 2013-08-10Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsKeywords: Administration, Intravenous ; Adult ; Animals ; Cytokines/immunology ; Female ; Humans ; Immunity, Cellular ; Malaria Vaccines/*administration & dosage/adverse effects/*immunology ; Malaria, Falciparum/*prevention & control ; Male ; Mice ; Plasmodium falciparum/*immunology ; Sporozoites/immunology ; T-Lymphocytes/immunology ; Vaccination/adverse effects/methodsPublished by: -
10Latest Papers from Table of Contents or Articles in PressL. Hunter ; J. Gordon ; S. Peck ; D. Ang ; J. F. Lin
American Association for the Advancement of Science (AAAS)
Published 2013Staff ViewPublication Date: 2013-02-23Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsPublished by: -
11Latest Papers from Table of Contents or Articles in PressJ. W. Moore ; C. Carr-Harris ; A. S. Gottesfeld ; D. MacIntyre ; D. Radies ; M. Cleveland ; C. Barnes ; W. Joseph ; G. Williams ; J. Gordon ; B. Shepert
American Association for the Advancement of Science (AAAS)
Published 2015Staff ViewPublication Date: 2015-08-08Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsKeywords: *Animal Migration ; Animals ; Canada ; Decision Making ; Fisheries ; *Oil and Gas Fields ; Risk ; *Rivers ; *SalmonPublished by: -
12Latest Papers from Table of Contents or Articles in PressL. L. Haak ; D. Baker ; D. K. Ginther ; G. J. Gordon ; M. A. Probus ; N. Kannankutty ; B. A. Weinberg
American Association for the Advancement of Science (AAAS)
Published 2012Staff ViewPublication Date: 2012-10-16Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsKeywords: Access to Information ; Data Mining ; Information Dissemination/*methods ; Internet/*standards ; Research Design/*standards ; Research Report/*standardsPublished by: -
13SSOARStaff View Fulltext
Publication Date: 2019-04-26Description: It is surprising that little research has been conducted by Eliasians on museums and that, with some exceptions, academics working on museums do not cite The Civilizing Process. All the more so given that: (i) museum research supports Elias’s claim that elements of modernity originated in court societies, (ii) the nineteenth-century museum was a leading edge of the West’s belief in itself as a singularly civilized place and (iii) there is a contradiction between the museum’s universalism and its latent capacity to stigmatize some visitors as uncivilized outsiders. Indeed, Elias’s theory of established-outsider relations offers profound insights into the museum dimension of social stigma and the socio-genesis of the museum. First, an Eliasian perspective illuminates the relationship between museums and the peculiar structures of feeling that flowed from the interdependencies of modernization. Secondly, in studying European upper classes, he stressed the co-existence of different propertied strata within nineteenth-century states. This explains the apparent inchoateness of European national museums as they emerged at the interface of ruling dynastic elites and upwardly mobile bourgeois outsiders. Thirdly, documentary evidence reveals the museum to be a place where middle class people incorporated and transformed a courtly habitus whilst simultaneously stigmatizing both aristocratic and working class ways of living the body. Finally, Elias elaborated dynamic models of established-outsider relations, emphasizing their ‘complex polyphony’ as the key to explaining the power to stigmatize. The museum performed that polyphony at the interface of established-outsider relations; it could be said that they were the very causes of museums.Keywords: Sociology & anthropology ; Soziologie, Anthropologie ; established-outsider relations ; Allgemeine Soziologie, Makrosoziologie, spezielle Theorien und Schulen, Entwicklung und Geschichte der Soziologie ; General Sociology, Basic Research, General Concepts and History of Sociology, Sociological Theories ; Elias, N. ; Klassenbewusstsein ; Klientelismus ; soziologische Theorie ; Moderne ; Adel ; Marginalität ; Besucher ; 19. Jahrhundert ; Museum ; Figuration ; soziale Klasse ; Stigmatisierung ; Bürgertum ; Modernisierung ; visitor ; modernization ; figuration ; aristocracy ; sociological theory ; social class ; stigmatization ; clientelism ; museum ; bourgeoisie ; class consciousness ; marginality ; modernity ; nineteenth century ; 10200Type: journal article, Zeitschriftenartikel -
14Articles: DFG German National LicensesTocher, Douglas R. ; Bell, J. Gordon ; Sargent, John R.
Oxford, UK : Blackwell Publishing Ltd
Published 1991Staff ViewISSN: 1471-4159Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: The incorporation of [3H]arachidonate ([3H]AA) and [14C]eicosapentaenoate ([14C]EPA) into glycerophos-pholipids was studied in isolated brain cells from rainbow trout, a teleost fish whose lipids are rich in (n-3) polyunsaturated fatty acids (PUF As), EPA was incorporated into total lipid to a greater extent than AA, but the incorporation of both PUF As into total glycerophospholipids was almost identical. The incorporation of both AA and EPA was greatest into phosphatidylethanolamine (PE). However, when expressed per milligram of individual phosphoglycerides, both AA and EPA were preferentially incorporated into phospha-tidylinositol (PI), the preference being significantly greater with AA. On the same basis, significantly more EPA than AA was incorporated into phosphatidylcholine (PC). When double-labelled cells were challenged with calcium ionophore A23187, the 3H and 14C released from the cells closely paralleled each other, peaking at 10 min after addition of ionophore.The 12-monohydroxylated derivative was the predominant lipoxygenase product from both AA and EPA with a rank order of 12-hydroxyeicosatetraenoic acid (12-HETE) 〉 leukotriene B4 (LTB4) 〉 5-HETE 〉 15-HETE for the AA products and 12-hydroxyeicosapentaenoic acid (12-HEPE) 〉 5-HEPE 〉 LTB5 〉 15-HEPE for EPA products. The 3H/14C (dpm/dpm) ratios in the glycerophospholipids, total released radioactivity, and the lipoxygenase products suggested that PC rather than PI was the likely source of eicosanoid precursors in trout brain cells.Type of Medium: Electronic ResourceURL: -
15Articles: DFG German National LicensesMartel, Cynthia L. ; Mackic, Jasmina B. ; Matsubara, Etsuro ; Governale, Samuel ; Miguel, Calero ; Miao, Wesley ; McComb, J. Gordon ; Frangione, Blas ; Ghiso, Jorge ; Zlokovic, Berislav V.
Oxford, UK : Blackwell Science Ltd
Published 1997Staff ViewISSN: 1471-4159Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Abstract: Cerebral capillary sequestration and blood-brain barrier (BBB) permeability to apolipoproteins E2 (apoE2), E3 (apoE3), and E4 (apoE4) and to their complexes with sAβ1–40, a peptide homologous to the major form of soluble Alzheimer's amyloid β, were studied in perfused guinea pig brain. Cerebrovascular uptake of three apoE isoforms was low, their blood-to-brain transport undetectable, but uptake by the choroid plexus significant. Binding of all three isoforms to sAβ1–40 in vitro was similar with a KD between 11.8 and 12.9 nM. Transport into brain parenchyma and sequestration by BBB and choroid plexus were negligible for sAβ1–40-apoE2 and sAβ1–40-apoE3, but significant for sAβ1–40-apoE4. After 10 min, 85% of sAβ1–40-apoE4 taken up at the BBB remained as intact complex, whereas free sAβ1–40 was 51% degraded. Circulating apoE isoforms have contrasting effects on cerebral capillary uptake of and BBB permeability of sAβ. ApoE2 and apoE3 completely prevent cerebral capillary sequestration and blood-to-brain transport of sAβ1–40. Conversely, apoE4, by entering brain microvessels and parenchyma as a stable complex with sAβ, reduces peptide degradation and may predispose to cerebrovascular and possibly enhance parenchymal amyloid formation under pathological conditions.Type of Medium: Electronic ResourceURL: -
16Articles: DFG German National LicensesSheu, Kwan-Fu Rex ; Calingasan, Noel Y. ; Lindsay, J. Gordon ; Gibson, Gary E.
Oxford, UK : Blackwell Science Ltd
Published 1998Staff ViewISSN: 1471-4159Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: Abstract: Mitochondrial dysfunction is a common feature of many neurodegenerative disorders. The metabolic encephalopathy caused by thiamine deficiency (TD) is a classic example in which an impairment of cerebral oxidative metabolism leads to selective cell death. In experimental TD in rodents, a reduction in the activity of the thiamine diphosphate-dependent, mitochondrial enzyme α-ketoglutarate dehydrogenase complex (KGDHC) occurs before the onset of pathologic lesions and is among the earliest biochemical deficits found. To understand the molecular basis and the significance of the deficiency of KGDHC in TD-induced brain damage, the enzyme activity and protein levels of KGDHC were analyzed. The effect of TD on the subregional/cellular distribution of KGDHC and the anatomic relation of KGDHC with selective cell death were also tested by immunocytochemistry. Consistent with several previous studies, TD dramatically reduced KGDHC activity in both anatomically damaged (thalamus and inferior colliculus) and spared (cerebral cortex) regions. Immunocytochemistry revealed no apparent correlation of regional KGDHC immunoreactivity or its response to TD with affected regions in TD. The basis of the enzymatic and immunocytochemical behavior of KGDHC was further assessed by quantitative immunoblots, using antibodies specific for each of the three KGDHC components. Despite the marked decrease of KGDHC activity in TD, no reduction of any of the three KGDHC protein levels was found. Thus, TD impairs the efficacy of the KGDHC catalytic machinery, whereas the concentration of protein molecules persists. The generalized decline of KGDHC activity with no apparent anatomic selectivity is consistent with the notion that the compromised mitochondrial oxidation sensitizes the brain cells to various other insults that precipitate the cell death. The current TD model provides a relevant experimental system to understand the molecular basis of many neurodegenerative conditions in which mitochondrial dysfunction and KGDHC deficiency are prominent features.Type of Medium: Electronic ResourceURL: -
17Articles: DFG German National LicensesGILBERT, JOHN C. ; ORTIZ, WINSTON R. ; MILLICHAP, J. GORDON
Oxford, UK : Blackwell Publishing Ltd
Published 1966Staff ViewISSN: 1471-4159Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineType of Medium: Electronic ResourceURL: -
18Articles: DFG German National LicensesALDERSON, SUSAN G. ; BARRATT, MARTIN D. ; BLACK, J. GORDON
Oxford, UK : Blackwell Publishing Ltd
Published 1984Staff ViewISSN: 1468-2494Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineNotes: The effect of aqueous solutions of 2-hydroxyacids of chain length C3 to C10 on the extensibility of undamaged and solvent-damaged guinea-pig footpad stratum corneum has been studied. The increase in extensibility of solvent-damaged corneum, caused by treatment with hydroxyacid, reached a maximum with 2-hydroxycaprylic acid (C8); on undamaged corneum, 2-hydroxycaprylic acid was the only hydroxyacid studied to give a significant effect. The increase in corneum extensibility produced by 2-hydroxyacids decreases when the pH is raised from 3 to 4. This loss of effect correlates with the ionization of the hydroxyacid (pK˜ 3.85).The binding of radiolabelled 2-hydroxycaproic (C6) and 2-hydroxycaprylic acids to stratum corneum has been studied. 2-Hydroxycaprylic acid binds much more strongly than 2-hydroxycaproic acid, the difference in the binding being consistent with the hydrophobic binding energy of two methylene groups. Raising the pH above 3.5 results in a large decrease in the binding of 2-hydroxycaprylic acid in line with the corresponding reduction in extensibility.Treatment with 2-hydroxyacids results in a small increase in the water-binding capacity of solvent-damaged stratum corneum, but in a decrease in the water-binding capacity of undamaged stratum corneum. These data are discussed in terms of a possible mechanism for the plasticizing of stratum corneum. Effet des 2-hydroxyacides sur la couche cornée du coussin des pattes de cobayeType of Medium: Electronic ResourceURL: -
19Articles: DFG German National LicensesStaff View
ISSN: 1467-9310Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: EconomicsNotes: This note augments recent comments in this journal relating to the tendency for schedulers of R & D to consistently underestimate the time needed for project completion. One author postulated the existence of psychological time-horizons, varying by individual, as the determinant of the degree of underestimation. A little-known graphic technique, the program trend chart, appears to provide an empirical means of compensating for the individual tendency toward underestimation of project duration. The technique may also provide a basis for measurement of an individual's time-horizon, through observation of the variation between actual and predicted completion dates.Type of Medium: Electronic ResourceURL: -
20Articles: DFG German National LicensesParish, Lawrence Charles ; Witkowski, Joseph A. ; Muir, J. Gordon
Oxford, UK : Blackwell Publishing Ltd
Published 1985Staff ViewISSN: 1365-4632Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: MedicineType of Medium: Electronic ResourceURL: