Search Results - (Author, Cooperation:H. Nishina)
-
1S. Porazinski ; H. Wang ; Y. Asaoka ; M. Behrndt ; T. Miyamoto ; H. Morita ; S. Hata ; T. Sasaki ; S. F. Krens ; Y. Osada ; S. Asaka ; A. Momoi ; S. Linton ; J. B. Miesfeld ; B. A. Link ; T. Senga ; A. Castillo-Morales ; A. O. Urrutia ; N. Shimizu ; H. Nagase ; S. Matsuura ; S. Bagby ; H. Kondoh ; H. Nishina ; C. P. Heisenberg ; M. Furutani-Seiki
Nature Publishing Group (NPG)
Published 2015Staff ViewPublication Date: 2015-03-18Publisher: Nature Publishing Group (NPG)Print ISSN: 0028-0836Electronic ISSN: 1476-4687Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsKeywords: Actomyosin/metabolism ; Adaptor Proteins, Signal Transducing/genetics/metabolism ; Animals ; Body Size/*genetics ; Embryo, Nonmammalian/anatomy & histology/embryology/metabolism ; Fish Proteins/genetics/*metabolism ; GTPase-Activating Proteins/metabolism ; Genes, Essential/genetics ; Gravitation ; Humans ; Morphogenesis/*genetics ; Mutation/genetics ; Organ Size/genetics ; Oryzias/*anatomy & histology/*embryology/genetics ; Phenotype ; Protein-Serine-Threonine Kinases/genetics/metabolism ; Signal Transduction ; Spheroids, Cellular/cytology/metabolismPublished by: -
2Goto, H., Nishio, M., To, Y., Oishi, T., Miyachi, Y., Maehama, T., Nishina, H., Akiyama, H., Mak, T. W., Makii, Y., Saito, T., Yasoda, A., Tsumaki, N., Suzuki, A.
The Company of Biologists
Published 2018Staff ViewPublication Date: 2018-03-29Publisher: The Company of BiologistsPrint ISSN: 0950-1991Electronic ISSN: 1477-9129Topics: BiologyKeywords: Musculoskeletal systemPublished by: -
3Nishina, H. ; Inageda, K. ; Takahashi, K. ; Hoshino, S. ; Ikeda, K. ; Katada, T.
Amsterdam : ElsevierStaff ViewISSN: 0006-291XSource: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyChemistry and PharmacologyPhysicsType of Medium: Electronic ResourceURL: -
4Staff View
ISSN: 0016-6480Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyMedicineType of Medium: Electronic ResourceURL: -
5Staff View
ISSN: 0021-9673Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: Chemistry and PharmacologyType of Medium: Electronic ResourceURL: -
6Staff View
ISSN: 0006-291XSource: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyChemistry and PharmacologyPhysicsType of Medium: Electronic ResourceURL: -
7Tanabe, H. ; Kumagai, N. ; Tsukahara, T. ; Ishiura, S. ; Kominami, E. ; Nishina, H. ; Sugita, H.
Amsterdam : ElsevierStaff ViewISSN: 0167-4889Keywords: (Rat keratinocyte) ; Cathepsin ; Differentiation ; Lysosomal proteinaseSource: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyChemistry and PharmacologyMedicinePhysicsType of Medium: Electronic ResourceURL: -
8Staff View
ISSN: 0304-8853Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: PhysicsType of Medium: Electronic ResourceURL: -
9Komine, M. ; Watabe, Y. ; Shimaoka, S. ; Sato, F. ; Kake, K. ; Nishina, H. ; Ohtsuki, M. ; Nakagawa, H. ; Tamaki, K.
Springer
Published 1999Staff ViewISSN: 1432-069XKeywords: Key words Vitamin D3 ; OCT ; Cytokines ; Nuclear factorsSource: Springer Online Journal Archives 1860-2000Topics: MedicineNotes: Abstract Topical vitamin D3 has relatively recently been introduced for the treatment of psoriasis. Synthetic vitamin D3 analogues with a high potential for inducing differentiation of cells, but with a low hypercalcemic effect have recently been developed. One such synthetic analogue of 1,25-dihydroxyvitamin D3 (calcitriol), 22-oxacalcitriol (OCT), is a novel agent for the topical treatment of psoriasis. The activity of OCT in vitro was investigated and compared with that of a series of vitamin D3 analogues as to their ability to inhibit murine T lymphocyte proliferation stimulated by con-A, to suppress IL-6 and IL-8 production by keratinocytes stimulated with IL-1α and TNFα, and to inhibit AP-1- and NFκB-dependent reporter gene expression. OCT inhibited the proliferation of lymphocytes and suppressed IL-8 and IL-6 production by keratinocytes to the same extent as the other vitamin D3 analogues. It also inhibited AP-1- and NFκB-controlled luciferase activity to the same extent as the other vitamin D3 analogues, which demonstrates its mechanism of action in the suppression of inflammatory processes.Type of Medium: Electronic ResourceURL: