Search Results - (Author, Cooperation:G. Stanton)
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1Latest Papers from Table of Contents or Articles in PressStaff View
Publication Date: 2018-05-17Publisher: American Physical Society (APS)Electronic ISSN: 2160-3308Topics: PhysicsPublished by: -
2Latest Papers from Table of Contents or Articles in PressStaff View
Publication Date: 2018-10-11Publisher: Institute of Physics (IOP)Electronic ISSN: 1367-2630Topics: PhysicsPublished by: -
3Latest Papers from Table of Contents or Articles in PressJ. N. Coleman ; M. Lotya ; A. O'Neill ; S. D. Bergin ; P. J. King ; U. Khan ; K. Young ; A. Gaucher ; S. De ; R. J. Smith ; I. V. Shvets ; S. K. Arora ; G. Stanton ; H. Y. Kim ; K. Lee ; G. T. Kim ; G. S. Duesberg ; T. Hallam ; J. J. Boland ; J. J. Wang ; J. F. Donegan ; J. C. Grunlan ; G. Moriarty ; A. Shmeliov ; R. J. Nicholls ; J. M. Perkins ; E. M. Grieveson ; K. Theuwissen ; D. W. McComb ; P. D. Nellist ; V. Nicolosi
American Association for the Advancement of Science (AAAS)
Published 2011Staff ViewPublication Date: 2011-02-05Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsPublished by: -
4Articles: DFG German National LicensesStaff View
ISSN: 1467-9310Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: EconomicsNotes: Technology, by its very nature, creates a high degree of interdependence in the industrial marketplace. Customers buy products for use with other products in a production system. The consequent requirement for technical compatibility constrains managers' freedom in developing products and marketing programmes. lndustrywide product standards, and the process of setting them, play a complex and little discussed role in enabling managers to deal with these constraints. This article provides a market-based examination of product standards and that role.Type of Medium: Electronic ResourceURL: -
5Articles: DFG German National LicensesStaff View
ISSN: 1749-6632Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: Natural Sciences in GeneralType of Medium: Electronic ResourceURL: -
6Articles: DFG German National LicensesStaff View
ISSN: 0022-5185Topics: Theology and Religious StudiesNotes: AUTHORS AND BOOKS REVIEWED OR NOTICEDURL: -
7Articles: DFG German National LicensesStaff View
ISSN: 0022-5185Topics: Theology and Religious StudiesNotes: AUTHORS AND BOOKS REVIEWED OR NOTICEDURL: -
8Articles: DFG German National LicensesStaff View
ISSN: 0022-5185Topics: Theology and Religious StudiesNotes: AUTHORS AND BOOKS REVIEWED OR NOTICEDURL: -
9Articles: DFG German National LicensesStaff View
ISSN: 0022-5185Topics: Theology and Religious StudiesNotes: AUTHORS AND BOOKS REVIEWED OR NOTICEDURL: -
10Articles: DFG German National LicensesStaff View
ISSN: 0022-5185Topics: Theology and Religious StudiesNotes: AUTHORS AND BOOKS REVIEWED OR NOTICEDURL: -
11Articles: DFG German National LicensesStaff View
ISSN: 0022-2437Topics: EconomicsURL: -
12Articles: DFG German National LicensesStaff View
ISSN: 0026-7074Topics: Classical StudiesURL: -
13Articles: DFG German National LicensesBaron, Samuel ; Blalock, J. Edwin ; Dianzani, Ferdinando ; Fleischmann, W. Robert ; Georgiades, Jerzy A. ; Johnson, Howard M. ; Stanton, G. John
Oxford, UK : Blackwell Publishing Ltd
Published 1980Staff ViewISSN: 1749-6632Source: Blackwell Publishing Journal Backfiles 1879-2005Topics: Natural Sciences in GeneralType of Medium: Electronic ResourceURL: -
14Articles: DFG German National LicensesStaff View
ISSN: 0018-0777Topics: Classical StudiesNotes: MISZELLENURL: -
15Articles: DFG German National LicensesStaff View
ISSN: 0018-0777Topics: Classical StudiesNotes: MISZELLENURL: -
16Articles: DFG German National LicensesStaff View
ISSN: 0018-0777Topics: Classical StudiesURL: -
17Articles: DFG German National LicensesStaff View
ISSN: 1432-0851Keywords: Melanoma ; Antitumor response ; Interferon resistance ; rHuIFNαA/DSource: Springer Online Journal Archives 1860-2000Topics: MedicineNotes: Abstract Mouse B16 melanoma cells rapidly develop resistance to the antiproliferative effects of interferon α (IFNα) and interferon β (IFNβ) when they are exposed to the interferons in vitro. This resistance was characterized to be non-genetic and dose-dependent, and does not alter other IFN-induced effects such as antiviral effects and elevation of 2′,5′-oligoadenylate synthetase activity in IFN-treated cells. The study of these IFN-resistant cells has been extended to an in vivo tumor model. Resistance, if it occurred in vivo, did not adversely affect the survival of IFN-treated mice. Further, IFN-treated mice inoculated with B16 cells that were resistant in vitro (B16αres cells) survived significantly longer than IFN-treated mice inoculated with B16 cells that were sensitive in vitro. The IFN-treated B16αres-inoculated mice had a significantly higher cure rate as well. The prolonged survival of the mice bearing B16αres cell tumors did not seem to be caused by the slower growth rate of the B16αres cells, since experiments performed with a tenfold higher B16αres cell inoculum and a tenfold lower B16 cell inoculum did not show any change in the survival pattern. It is clear that in vitro resistant B16αres cells are more sensitive to antitumor effects induced by IFN in vivo than in vitro sensitive B16 cells.Type of Medium: Electronic ResourceURL: -
18Articles: DFG German National LicensesStaff View
ISSN: 1432-0851Keywords: Key words: Melanoma – Antitumor response – Interferon resistance – rHuIFNαA/DSource: Springer Online Journal Archives 1860-2000Topics: MedicineNotes: Abstract. Mouse B16 melanoma cells rapidly develop resistance to the antiproliferative effects of interferon α (IFNα) and interferon β (IFNβ) when they are exposed to the interferons in vitro. This resistance was characterized to be non-genetic and dose-dependent, and does not alter other IFN-induced effects such as antiviral effects and elevation of 2′,5′-oligoadenylate synthetase activity in IFN-treated cells. The study of these IFN-resistant cells has been extended to an in vivo tumor model. Resistance, if it occurred in vivo, did not adversely affect the survival of IFN-treated mice. Further, IFN-treated mice inoculated with B16 cells that were resistant in vitro (B16αres cells) survived significantly longer than IFN-treated mice inoculated with B16 cells that were sensitive in vitro. The IFN-treated B16αres-inoculated mice had a significantly higher cure rate as well. The prolonged survival of the mice bearing B16αres cell tumors did not seem to be caused by the slower growth rate of the B16αres cells, since experiments performed with a tenfold higher B16αres cell inoculum and a tenfold lower B16 cell inoculum did not show any change in the survival pattern. It is clear that in vitro resistant B16αres cells are more sensitive to antitumor effects induced by IFN in vivo than in vitro sensitive B16 cells.Type of Medium: Electronic ResourceURL: -
19Articles: DFG German National LicensesStaff View
ISSN: 0278-6656Topics: Classical StudiesURL: -
20Articles: DFG German National LicensesStaff View
ISSN: 0022-5185Topics: Theology and Religious StudiesNotes: AUTHORS AND BOOKS REVIEWED OR NOTICEDURL: