Search Results - (Author, Cooperation:D. Medina)
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1Latest Papers from Table of Contents or Articles in PressTorben Sick, Alexander G. Hufnagel, Jonathan Kampmann, Ilina Kondofersky, Mona Calik, Julian M. Rotter, Austin Evans, Markus Döblinger, Simon Herbert, Kristina Peters, Daniel Böhm, Paul Knochel, Dana D. Medina, Dina Fattakhova-Rohlfing and Thomas Bein
American Chemical Society (ACS)
Published 2018Staff ViewPublication Date: 2018-01-31Publisher: American Chemical Society (ACS)Print ISSN: 0002-7863Electronic ISSN: 1520-5126Topics: Chemistry and PharmacologyPublished by: -
2Latest Papers from Table of Contents or Articles in PressDossa, R. G., Cunningham, T., Sommermeyer, D., Medina-Rodriguez, I., Biernacki, M. A., Foster, K., Bleakley, M.
American Society of Hematology (ASH)
Published 2018Staff ViewPublication Date: 2018-01-05Publisher: American Society of Hematology (ASH)Print ISSN: 0006-4971Electronic ISSN: 1528-0020Topics: BiologyMedicineKeywords: Immunobiology and Immunotherapy, Transplantation, Myeloid Neoplasia, Lymphoid Neoplasia, Clinical Trials and ObservationsPublished by: -
3Latest Papers from Table of Contents or Articles in PressC. Settembre ; C. Di Malta ; V. A. Polito ; M. Garcia Arencibia ; F. Vetrini ; S. Erdin ; S. U. Erdin ; T. Huynh ; D. Medina ; P. Colella ; M. Sardiello ; D. C. Rubinsztein ; A. Ballabio
American Association for the Advancement of Science (AAAS)
Published 2011Staff ViewPublication Date: 2011-05-28Publisher: American Association for the Advancement of Science (AAAS)Print ISSN: 0036-8075Electronic ISSN: 1095-9203Topics: BiologyChemistry and PharmacologyComputer ScienceMedicineNatural Sciences in GeneralPhysicsKeywords: Active Transport, Cell Nucleus ; Animals ; *Autophagy ; Basic Helix-Loop-Helix Leucine Zipper Transcription Factors/genetics/*metabolism ; COS Cells ; Cell Nucleus/*metabolism ; Cells, Cultured ; Cercopithecus aethiops ; Cytoplasm/metabolism ; Gene Expression Regulation ; HeLa Cells ; Humans ; Liver/metabolism ; Lysosomes/*metabolism ; MAP Kinase Signaling System ; Mice ; Mice, Transgenic ; Microtubule-Associated Proteins/metabolism ; Mitogen-Activated Protein Kinase 1/metabolism ; Phagosomes/metabolism ; Phosphorylation ; RNA Interference ; Transcription, Genetic ; Up-RegulationPublished by: -
4Latest Papers from Table of Contents or Articles in PressErika Virmani, Julian M. Rotter, Andre Mähringer, Tobias von Zons, Adelheid Godt, Thomas Bein, Stefan Wuttke, Dana D. Medina
American Chemical Society (ACS)
Published 2018Staff ViewPublication Date: 2018-03-30Publisher: American Chemical Society (ACS)Print ISSN: 0002-7863Electronic ISSN: 1520-5126Topics: Chemistry and PharmacologyPublished by: -
5Latest Papers from Table of Contents or Articles in PressErber, A. C., Arana, B., Bennis, I., Ben Salah, A., Boukthir, A., Castro Noriega, M. d. M., Cisse, M., Cota, G. F., Handjani, F., Kebede, M. G., Lang, T., Lopez Carvajal, L., Marsh, K., Martinez Medina, D., Plugge, E., Olliaro, P.
BMJ Publishing
Published 2018Staff ViewPublication Date: 2018-06-17Publisher: BMJ PublishingElectronic ISSN: 2044-6055Topics: MedicineKeywords: Open access, Qualitative researchPublished by: -
6Articles: DFG German National LicensesStaff View
ISSN: 0006-291XSource: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyChemistry and PharmacologyPhysicsType of Medium: Electronic ResourceURL: -
7Articles: DFG German National LicensesStaff View
ISSN: 0012-1606Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyType of Medium: Electronic ResourceURL: -
8Articles: DFG German National LicensesStaff View
ISSN: 0014-4827Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyMedicineType of Medium: Electronic ResourceURL: -
9Articles: DFG German National LicensesStaff View
ISSN: 0014-4827Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyMedicineType of Medium: Electronic ResourceURL: -
10Articles: DFG German National LicensesStaff View
ISSN: 0531-5565Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: MedicineType of Medium: Electronic ResourceURL: -
11Articles: DFG German National LicensesStaff View
ISSN: 0531-5565Source: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: MedicineType of Medium: Electronic ResourceURL: -
12Articles: DFG German National LicensesStaff View
ISSN: 0006-291XSource: Elsevier Journal Backfiles on ScienceDirect 1907 - 2002Topics: BiologyChemistry and PharmacologyPhysicsType of Medium: Electronic ResourceURL: -
13Articles: DFG German National LicensesStaff View
ISSN: 1435-1463Keywords: Dopamine ; deprenyl ; methamphetamine ; Parkinson's diseaseSource: Springer Online Journal Archives 1860-2000Topics: MedicineNotes: Summary The effects of deprenyl on methamphetamine-induced dopamine depletions were studied in mice. Four SC injections of 12.5 mg/kg of methamphetamine at two-hour intervals caused substantial (72–82%) and longlasting depletions of striatal dopamine. Pretreatment with either 25 or 40 mg/ kg of deprenyl did not significantly alter the magnitude of this depletion. These results indicate that, unlike what is observed following MPTP, there is no protection afforded dopaminergic cells by deprenyl pretreatment in the methamphetamine model of parkinsonism.Type of Medium: Electronic ResourceURL: -
14Articles: DFG German National LicensesStaff View
ISSN: 1476-4687Source: Nature Archives 1869 - 2009Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsNotes: [Auszug] We have tested the cell-mediated immune stimulation hypothesis directly by using a microcytotoxicity test, which is very sensitive for measuring the effects of lymphocytes and serum from sensitized mice on tumour growth, and has been described by Hellstrom and Hellstrom5. Briefly, tumour cells from ...Type of Medium: Electronic ResourceURL: -
15Articles: DFG German National LicensesStaff View
ISSN: 1432-0800Source: Springer Online Journal Archives 1860-2000Topics: Energy, Environment Protection, Nuclear Power EngineeringMedicineType of Medium: Electronic ResourceURL: -
16Articles: DFG German National LicensesStaff View
ISSN: 1615-6102Keywords: Stem cells ; Phenotypic markers ; Proliferation potential ; Keratins ; Basal cells ; Preneoplasias ; DevelopmentSource: Springer Online Journal Archives 1860-2000Topics: BiologyNotes: Summary The evidence for mammary epithelial stem cells and their phenotypic characteristics in normal and neoplastic development is reviewed. The presence of stem cells in all parts of the mammary parenchyma at all stages of differentiation has been demonstrated by transplantation experiments. The phenotypic characterization of stem cells has been defined by a battery of monospecific antibodies. These studies suggest that a mammary epithelium stem cell compartment exists in the basal layer of the gland as well as in the end bud. Whether these same stem cells are expressed in mammary preneoplasias and neoplasias has not been answered conclusively. Phenotypic markers specific for stem cells and stably expressed in transformed cell populations are needed to follow the fate of stem cells.Type of Medium: Electronic ResourceURL: -
17Articles: DFG German National LicensesThompson, M. P. ; Farrell, H. M. ; Mohanam, Sanjeeva ; Liu, Sue ; Kidwell, W. R. ; Bansal, M. P. ; Cook, R. G. ; Medina, D. ; Kotts, Claire E. ; Bano, Mozeena
Springer
Published 1992Staff ViewISSN: 1615-6102Keywords: Milk protein ; Mammary cells ; Cell growth ; InhibitionSource: Springer Online Journal Archives 1860-2000Topics: BiologyNotes: Summary A growth inhibitory protein, mammary inhibitory activity (MIA), was purified to apparent homogeneity from human milk. At concentrations of 5 to 10 ng/ml, the factor inhibited the growth of mammary epithelial cells by 30–80% and also inhibited the growth of normal rat kidney cells. Whereas the cell division of normal human mammary epithelium in primary culture was inhibited by MIA, cell division by fibroblasts from the same tissues was unresponsive. Inhibition was dose and time dependent and readily reversed when MIA was removed. MIA also inhibited growth in culture for three cell lines. The growth inhibitory protein migrated as a 14 kDa protein under reducing conditions on polyacrylamide gels in the presence of sodium dodecyl sulfate. The apparent isoelectric point was pI 5.0. The amino acid composition of MIA resembled that of α-lactalbumin, and sequence analysis of the N-terminal region comprising residues 1–24 and an isolated peptide were identical with the N-terminal and residues 66–81 of human α-lactalbumin. In addition, MIA was active in the lactose synthase system. The results strongly suggest that MIA and α-lactalbumin are identical proteins. Consistent with these results, α-lactalbumin preparations from several mammalian species, including human, goat, cow and camel, were all found to be growth inhibitory for cultured mammary epithelial cells. The inhibitory activity associated with human α-lactalbumin was destroyed by digestion with pepsin or chymotrypsin, by carboxymethylation of cysteine, or by cleavage of methionine 90 following cyanogen bromide treatment. The results raise the possibility that during lactation α-lactalbumin, a product of mammary cell differentiation, could be a physiologically relevant feed-back inhibitor of mammary cell growth and perhaps of other cell types as well.Type of Medium: Electronic ResourceURL: -
18Articles: DFG German National LicensesStaff View
ISSN: 0748-8025Keywords: Engineering ; Engineering GeneralSource: Wiley InterScience Backfile Collection 1832-2000Topics: MathematicsTechnologyAdditional Material: 3 Ill.Type of Medium: Electronic ResourceURL: