Search Results - (Author, Cooperation:C. I. Newbold)
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1Latest Papers from Table of Contents or Articles in PressM. Manske ; O. Miotto ; S. Campino ; S. Auburn ; J. Almagro-Garcia ; G. Maslen ; J. O'Brien ; A. Djimde ; O. Doumbo ; I. Zongo ; J. B. Ouedraogo ; P. Michon ; I. Mueller ; P. Siba ; A. Nzila ; S. Borrmann ; S. M. Kiara ; K. Marsh ; H. Jiang ; X. Z. Su ; C. Amaratunga ; R. Fairhurst ; D. Socheat ; F. Nosten ; M. Imwong ; N. J. White ; M. Sanders ; E. Anastasi ; D. Alcock ; E. Drury ; S. Oyola ; M. A. Quail ; D. J. Turner ; V. Ruano-Rubio ; D. Jyothi ; L. Amenga-Etego ; C. Hubbart ; A. Jeffreys ; K. Rowlands ; C. Sutherland ; C. Roper ; V. Mangano ; D. Modiano ; J. C. Tan ; M. T. Ferdig ; A. Amambua-Ngwa ; D. J. Conway ; S. Takala-Harrison ; C. V. Plowe ; J. C. Rayner ; K. A. Rockett ; T. G. Clark ; C. I. Newbold ; M. Berriman ; B. MacInnis ; D. P. Kwiatkowski
Nature Publishing Group (NPG)
Published 2012Staff ViewPublication Date: 2012-06-23Publisher: Nature Publishing Group (NPG)Print ISSN: 0028-0836Electronic ISSN: 1476-4687Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsKeywords: Alleles ; *Biodiversity ; Genome, Protozoan ; Genotype ; *High-Throughput Nucleotide Sequencing ; Humans ; Malaria, Falciparum/*parasitology ; Phylogeny ; Plasmodium falciparum/classification/*genetics ; Polymorphism, Single Nucleotide ; Principal Component AnalysisPublished by: -
2Articles: DFG German National LicensesRuwende, C. ; Khoo, S. C. ; Snow, R. W. ; Yates, S. N. R. ; Kwiatkowski, D. ; Gupta, S. ; Warn, P. ; Allsopp, C. E. M. ; Gilbert, S. C. ; Peschu, N. ; Newbold, C. I. ; Greenwood, B. M. ; Marsh, K. ; Hill, A. V. S.
[s.l.] : Nature Publishing Group
Published 1995Staff ViewISSN: 1476-4687Source: Nature Archives 1869 - 2009Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsNotes: [Auszug] Despite the extensive heterogeneity of G6PD deficiency, a single molecular variant, G6PD A-, seems predominant in sub-Saharan Africa12 14. It differs from the normal G6PD B allele at two amino-acid positions, one at position 376 encoding the B to A change and the other at position 202 that ...Type of Medium: Electronic ResourceURL: -
3Articles: DFG German National LicensesIntercellular adhesion molecule-1 is an endothelial cell adhesion receptor for Plasmodium falciparumBerendt, A. R. ; Simmons, D. L. ; Tansey, J. ; Newbold, C. I. ; Marsh, K.
[s.l.] : Nature Publishing Group
Published 1989Staff ViewISSN: 1476-4687Source: Nature Archives 1869 - 2009Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsNotes: [Auszug] We have observed that binding of red blood cells infected with Gambian wild isolates of P. falciparum, in the C32 amelanotic melanoma cell adherence assay varies markedly, but does not correlate with severity of clinical malaria11. More recently we have found that there is no correlation ...Type of Medium: Electronic ResourceURL: -
4Articles: DFG German National LicensesRobson, Kathryn J. H. ; Hall, Jennifer R. S. ; Jennings, M. W. ; Harris, T. J. R. ; Marsh, K. ; Newbold, C. I. ; Tate, Valerie E. ; Weatherall, D. J.
[s.l.] : Nature Publishing Group
Published 1988Staff ViewISSN: 1476-4687Source: Nature Archives 1869 - 2009Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsNotes: [Auszug] The human malaria parasite, P. falciparum follows a complex life-cycle, during which different antigens are expressed at particular developmental stages. The major antigen on the sporozoite surface is the CS protein, which probably determines the specificity of the interaction between the parasite ...Type of Medium: Electronic ResourceURL: -
5Articles: DFG German National LicensesWilliams, T. N. ; Maitland, K. ; Bennett, S. ; Ganczakowski, M. ; Peto, T. E. A. ; Newbold, C. I. ; Bowden, D. K. ; Weatherall, D. J. ; Clegg, J. B.
[s.l.] : Nature Publishing Group
Published 1996Staff ViewISSN: 1476-4687Source: Nature Archives 1869 - 2009Topics: BiologyChemistry and PharmacologyMedicineNatural Sciences in GeneralPhysicsNotes: [Auszug] The a+-thalassaemias are usually caused by the deletion of one of the linked pair of a-globin genes on chromosome 16 (ref. 5). The normal genotype is represented by aa/aa. Heterozygotes (á€"a/aa) are clinically normal, whereas homozygotes (á€"a/- a) have a mild ...Type of Medium: Electronic ResourceURL: